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45-Year-Old Female with Motor Nerve Damage
In Chapters 7 and 8, you met Kayla Tanner, who suffered a dislocated right hip in a car accident. Six weeks later, Mrs. Tanner was still unable to walk or run without hip pain, and had weakness in flexion at the knee, excessive foot inversion and plantar flexion, and significant “foot drop” (the inability to dorsiflex the foot).
Electromyography (measurement of muscle electrical activity) and nerve conduction studies (measurement of the speed of nerve impulses) revealed sciatic nerve damage, most likely a result of the nerve being compressed when the hip was dislocated. This large nerve innervates many lower limb muscles. Since her surgery, Mrs. Tanner has been undergoing intense physical therapy and has shown significant improvement.
6. + NCLEX-Style A muscle that originates on the posterior surface of the ischium and inserts on the posterior surface of the tibia will:
a. Flex the thigh at the hip
b. Extend the thigh at the hip
c. Flex the leg at the knee
d. Extend the leg at the knee
e. Flex the thigh and extend the leg
f. Flex the leg and extend the thigh
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Chapter 10 Solutions
HUMAN ANATOMY+PHYSIOLOGY-PKG.
- "One of the symmetry breaking events in mouse gastrulation requires the amplification of Nodal on the side of the embryo opposite to the Anterior Visceral Endoderm (AVE). Describe one way by which Nodal gets amplified in this region." My understanding of this is that there are a few ways nodal is amplified though I'm not sure if this is specifically occurs on the opposite side of the AVE. 1. pronodal cleaved by protease -> active nodal 2. Nodal -> BMP4 -> Wnt-> nodal 3. Nodal-> Nodal, Fox1 binding site 4. BMP4 on outside-> nodal Are all of these occuring opposite to AVE?arrow_forwardIf four babies are born on a given day What is the chance all four will be girls? Use genetics lawsarrow_forwardExplain each punnet square results (genotypes and probabilities)arrow_forward
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- Plekhg5 functions in bottle cell formation, and Shroom3 functions in neural plate closure, yet the phenotype of injecting mRNA of each into the animal pole of a fertilized egg is very similar. What is the phenotype, and why is the phenotype so similar? Is the phenotype going to be that there is a disruption of the formation of the neural tube for both of these because bottle cell formation is necessary for the neural plate to fold in forming the neural tube and Shroom3 is further needed to close the neural plate? So since both Plekhg5 and Shroom3 are used in forming the neural tube, injecting the mRNA will just lead to neural tube deformity?arrow_forwardWhat are some medical issues or health trends that may have a direct link to the idea of keeping fat out of diets?arrow_forwardwhat did charles darwin do in sciencearrow_forward
- Medical Terminology for Health Professions, Spira...Health & NutritionISBN:9781305634350Author:Ann Ehrlich, Carol L. Schroeder, Laura Ehrlich, Katrina A. SchroederPublisher:Cengage LearningEssentials of Pharmacology for Health ProfessionsNursingISBN:9781305441620Author:WOODROWPublisher:Cengage
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