You spray an insect with common insecticide that destroys the ability of acetylcholinesterase to recycle acetylcholine. What thenhappens?O The lack of recycled acetylcholine brings the cell metabolism to a halt.The insect loses control of body functions as nerve impulses flow continuously AND this prevents the synapse from restoringits ability to "reset" itself for the next impulse.O This kills the neuron directly.O This prevents the synapse from restoring its ability to "reset" itself for the next impulse.O The insect loses control of body functions as nerve impulses flow continuously.+40+20-31--20-40E-65145 6In Figure 17.1, number 5 representsO threshold.O action potential.resting potential.O depolarization.O repolarization.4.2.Membrane Potential (mV) A post-synaptic membraneO must be cell body membrane.O must be dendrite membrane.O can be either axon or cell body membrane.O can be either axon or dendrite membrane.O must be axon membrane.Neurotransmitters are molecules that cross the synaptic cleft andO either excite or inhibit the postsynaptic neuron.O always inhibit the postsynaptic neuron.O integrate the presynaptic action potential.O are carried along the membrane surface of the next neuron.O always excite the postsynaptic neuron.The likely effect on a neuron of two excitatory signals and twenty inhibitory signals isO confused integration.O transmission of a nerve impulse releasing excitatory neurotransmitters at the next synapse.O prohibiting the axon from firing at all.O transmission of a nerve impulse.O transmission of a nerve impulse releasing inhibitory neurotransmitters at the next synapse.

Disclaimer: Since you have asked multiple question, we will solve the first question for you. If you…

You spray an insect with common insecticide that destroys the ability of acetylcholinesterase to recycle acetylcholine. What then happens? O The lack of recycled acetylcholine brings the cell metabolism to a halt. The insect loses control of body functions as nerve impulses flow continuously AND this prevents the synapse from restoring its ability to "reset" itself for the next impulse. O This kills the neuron directly. O This prevents the synapse from restoring its ability to "reset" itself for the next impulse. O The insect loses control of body functions as nerve impulses flow continuously.

You spray an insect with common insecticide that destroys the ability of acetylcholinesterase to recycle acetylcholine. What then happens? O The lack of recycled acetylcholine brings the cell metabolism to a halt. The insect loses control of body functions as nerve impulses flow continuously AND this prevents the synapse from restoring its ability to "reset" itself for the next impulse. O This kills the neuron directly. O This prevents the synapse from restoring its ability to "reset" itself for the next impulse. O The insect loses control of body functions as nerve impulses flow continuously.

Human Anatomy & Physiology (11th Edition)

11th Edition

ISBN:9780134580999

Author:Elaine N. Marieb, Katja N. Hoehn

Publisher:Elaine N. Marieb, Katja N. Hoehn

Chapter1: The Human Body: An Orientation

Section: Chapter Questions

Problem 1RQ: The correct sequence of levels forming the structural hierarchy is A. (a) organ, organ system,...

See similar textbooks

Similar questions

Organophosphate pesticides inhibit the action of acetylcholinesterase. What effect does this have in the cell? Select one: a. It prevents synthesis of ACh in the presynaptic terminal causing less neurotransmitter release b. It prevents breakdown of ACh in the synapse causing increased activity at the postsynaptic cell c. It prevents reuptake of ACh into the presynaptic terminal causing increased activity at the postsynaptic cell d. It prevents binding of ACh on the postsynaptic cell causing less activity e. It prevents reuptake of ACh into the presynaptic terminal causing less neurotransmitter synthesis.
Once an action potential reaches a neurotransmitter release site, the release of the neurotransmitter is triggered by: The influx of Na* through voltage-gated channels. O The efflux of Na* through voltage-gated channels. The influx of K* through voltage-gated channels. The efflux of K+ through voltage-gated channels. The influx of Ca²+ through voltage-gated channels. O The efflux of Ca2+ through voltage-gated channels.
are these True or False? Receptors on the post-synaptic cell membrane that bind the acetylcholine are voltage-gated channels (channels that open in response to a change in the electrical charge of the membrane). When a muscle cell is not contracting its cell membrane is negative on the inner surface. The effect of a neurotransmitter on the muscle cell membrane is to modify its ion permeability properties temporarily.
A theoretical drug blocks Na* leak channels - what effect would this have on a neuron's resting membrane potential and why? Would this drug affect the neuron's ability to generate an action potential and why/why not?
Conduction velocity refers to the [a] at which an action potential travels along a neuron's axon. In invertebrates, conduction velocity can only be increased by [b]. However in vertebrates (exclusively), such as humans, conduction velocity can also be increased by [c]. This enables vertebrates to conserve [d] while still increasing conduction velocity. [a] [b] [c] [d] [Choose ] [Choose ] [Choose ] [Choose ]
Which of the following statements is FALSE? (Choose the best answer) O Neurons in the nervous system use electrical and chemical synapses to communicate between neurons and their target cells. Chemical synapses allow presynaptic neurons to excite or inhibit the postsynaptic neurons. O Electrical synapses allow presynaptic neurons to excite or inhibit the postsynaptic neurons. O Electrical synapses allow direct exchanges of both electric current and cytoplasmic contents. A single neuron uses either the electrical or chemical synapses to communicate with its target cells.
What is the function of the axon? to receive information and information into the cell body O to store genetic material and act as the metabolic centre of the neuron O to transmit information away from the soma to other neurons O to separate one neuron from neighbouring neurons
Matching (may not use all choices; may use some choices more than once) 45. Sección Release of inhibitory neurotransmitter 46. Decreased concentration of leaky sodium channels in the membrane Arsenic poisoning prevents mitochondria from making ATP 47. 48. Extreme sodium deficiency 49. Acetylcholinesterase concentration is decreased 50. Overdose of Potassium Chloride 51. Increased concentration of leaky potassium channels in the membrane Salto de columna A. Makes RMP more negative B. Makes RMP less negative
In muscles, acetylcholine is released by the _________________________and opens specific channels that allow the ion ______ to depolarize the membrane. motor neuron, sodium motor neuron, potassium muscle fibril, sodium muscle fibril, potassium
Researchers have designed a neuron that's unable to produce and release neurotransmitters. Can that neuron still have an action potential? Choose the answer with the correct answer and the correct reasoning. O No, action potentials need neurotransmitters to increase message speed O No, a neuron can't receive messages without its own neurotransmitters O No, a lack of neurotransmitters would leave a neuron unmyelinated O No, action potentials don't occur in the axon O Yes, action potentials don't require neurotransmitters:
Acetylcholine is an excitatory neurotransmitter that is responsible for initiating muscle movement. Consider the scenario where a mouse was bitten by a snake that injected venom into their circulatory system. The venom has an inhibitory effect on the reuptake of acetylcholine from the synaptic cleft. Explain how the mouse’s body would respond to the venom as well as the venoms effect at the cellular level.
4.3. After the rising phase, which ion channel is responsible for action potential returning to its resting V and why? Na channels, because positive Na* ions are at higher concentration outside the axon than inside, and so they enter the axon and raise V O Na* channels, because positive Na* ions are at higher concentration inside the axon than outside, and so they leave the axon and raise V, m O K* channels, because positive K* ions are at higher concentration outside the axon than inside, and so they enter the axon and raise Vm U K* channels, because positive K* ions are at higher concentration inside the axon than outside, and so they leave the axon and lower Vm