What will be the product(s) of exhaustive methylation of lactosefollowed by cleavage?A. 1,2,3,4,6-penta-O methyl glucoseB. 1,2,3,6-tetra-O-methyl galactose and 2,3,4,6-tetra-O-methyl glucoseC. 1,2,3,4,6-penta-O-methyl galactoseD. 1,2,3-tri-O-methyl glucose, 1,2,3,4 tetra-O-methyl-galactose and 2,3,4,6-tetra-O-methyl glucoseE. 2,3-di-O-methyl galactose

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Biochemistry

9th Edition

ISBN:9781319114671

Author:Lubert Stryer, Jeremy M. Berg, John L. Tymoczko, Gregory J. Gatto Jr.

Publisher:Lubert Stryer, Jeremy M. Berg, John L. Tymoczko, Gregory J. Gatto Jr.

Chapter1: Biochemistry: An Evolving Science

Section: Chapter Questions

Problem 1P

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A mutation caused by a base deamination or a tautomerization is called a a. silent mutation b. transition mutation c. nonsense mutation d. missense mutation
The following statements apply to compounds to which HONO, benzo(a)pyrene, aflatoxin, formaldehyde and chloroform are examples of: A. All may alter DNA sequences because they alkylate nucleotide bases B. All may alter DNA sequences which could result in altered protein products. C. All are mutagenic because they intercalate DNA D. All are mutagenic and causes the production of apurinic sites O E. All are mutagenic because they cause substitution of a base resulting in the production of a protein product with altered sequence
The key regulatory enzyme in pyrimidine synthesis is: a. Aspartate transcarbamoylase. b. Glutamine phosphoribosyl amidotransferase. c. Uridine monophosphate synthetase. d. Orotidylate decarboxylase
Which of the following is NOT a probable mechanism related to the killing action of radiation (UV and ionizing) A. T-T cyclobutane dimer formation B. Interstrand cross-linking C. Sugar-phosphate backbone breaks D. Intrastrand cross-linking E. Production of apurinic sites
A mutation in which of these proteins will lead to increased mutations in all daughter cells? A. helicase B. topoisomerase C. ligase D. DNA polymerase
A genetic disease called glucose-6-phosphate dehydrogenase deficiency is inherited in a manner similar to that of sickle-cell anemia except that it occurs most frequently in males. The defective enzyme cannot keep erythrocytes supplied with sufficient amounts of the antioxidant molecule NADPH NADPH protects cell membranes and other cellular structures from oxidation. Describe in general terms the inheritance pattern of this molecular disease. Why do you think that the antimalarial drug primaquine, which stimulates peroxide formation, results in devastating cases of hemolytic anemia in carriers of the defective gene? Does it surprise you that this genetic anomaly is commonly found in African and Mediterranean populations?
In the synthesis of 1 mole 1-lauroyl-2-myristoyl glycerol, the number of moles of water released is? a. 1 b. 2 c. 3 d. none of the above e. 4 Are both statements true? 1. The carbon number in ribose where guanine is connected is 1. 2. The other name for unprocessed eukaryotic RNA is raw nuclear RNA.
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Give an example of a reducing sugar and a non-reducing sugar. What is the chemical group responsible for the reduction reaction in a reducing sugar? Certain fats are important components of a healthy diet. But transfats are associated with increased riskof heart disease. Are transfats saturated or unsaturated fatty acids? What is the difference in chemicalstructure between a transfat versus a cisfat? DNA and RNA are information molecules with different roles in gene expression. List three differences between these two types of molecules.
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what is the answer to this question? Is my answer correct? Please explain in detail.
Name the tissues that contained the highest concentration of peroxidase. 2. Describe one function that peroxidase may perform in each of these tissues. Name one plant tissue area that contained proteins that were transferred to the nitrocellulose but did not contain peroxidase. 1. 3.

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