TRUE OR FALSE 1. A knockout mouse ia a mouse wherein certain genes are overexpressed. 2. Human gut microbota are rarely used in metabolic studies due to their differences in metabolism. 3. Cancer cell lines are often drevied from actual clinical tumors.
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- Match the following. Write correct answer in the form of 1a, 5b, etc. in the space provided. 1. Gene Replacement A. No active gene is present 2. Gene Knockout B. Only mutant gene is active 3. Gene Addition C. Both normal and mutant genes are activeWrite TRUE or FALSE. If false, write the word/s that make(s) the statement incorrect. 1.Metabolic pathways can be switched on or off by hormones only. 2.In epigenetics, the chemical tag ethyl group can result in the inhibition of gene expression.Cell and Molecular Biology Subject: 1. Explain how the actions of the lac operon are different from the trp operon. 2. Why is it important for a multicellular organism to be able to turn on only some of its genes in any particular tissue or cell type?
- Induced pluripotent stem cells have allowed scientists to model various diseases and screen drugs in these in vitro models. Please provide information on induced pluripotent stem cells and their use as described above (model various diseases and screen drugs in these in vitro models). Please explain clearly and nicely. THANKSOO HUAWEI nova 2 Plus DUAL CAMERA Which of the following molecules is designed to deliver drugs to the body cells? O a. Proteasome Ob. Lysosome O C. Liposome O d. Electroporation O e. Lysozyme If you were studying the effect of the TCA epigenetic drug on the expression of a tumor suppressor gene, the most predicted result would be? Oa. The histone deacetylation will increase Ob. The methylation pattern will remain constant O C. The expression of the gene will increase O d. The methylation of the gene will be increased e. The expression of the gene will decrease 80 NEXT PAGEDescribe one chemotherapeutic approach that can be used to treat prostate cancer? Your answer should include information about the molecular processes and interactions which are at the core of the treatment strategy
- Which statements are true? Explain why or why not.1 The chemical carcinogen dimethylbenz[a]anthra-cene (DMBA) must be an extraordinarily specific mutagensince 90% of the skin tumors it causes have an A-to-T alter-ation at exactly the same site in the mutant Ras gene.2 In the cellular regulatory pathways that controlcell growth and proliferation, the products of oncogenesare stimulatory components and the products of tumorsuppressor genes are inhibitory components.3 Cancer therapies directed solely at killing the rap-idly dividing cells that make up the bulk of a tumor areunlikely to eliminate the cancer from many patients.4 The main environmental causes of cancer are theproducts of our highly industrialized way of life such aspollution and food additives.Looking at this statement, develop an argument and critically evaluate/discuss it, then discuss difficulties with PARP inhibitor therapy and developments/progression in the future, the statement is "New synthetic lethal strategies targeting DNA damage response in solid tumours have been developed as a result of the success of PARP inhibitor therapy in ovarian cancer.ABOUT Phenylketonuria Explain Potential technical issues and limitations of PCR technology are mentioned Correct information about tissue that can be used to test for a genetic disease and justification of tissue selection Detailed information about the position (exact base pair number) of the new mutation relative to the sequence of the PAH gene. Numbering is based on the start of transcription of the PAH gene. PLEASE ANSWER ALLLL PLEASEE
- NO answer needs more than 2 sentences, some require just a couple words. 7. GTP-y-s is an analog of GTP that cannot be hydrolyzed. How would you expect a Co-IP experiment between G-alpha and G-beta proteins to differ in the presence of GTP-y-a and GDP? 8. An epithelial cell line expressed a fluorescence-based cAMP biosensor in all cells, but only expressed a GPCR in ~30% of those cells. When you treated the cells with the GPCR ligand, fluorescence from the cAMP sensor immediately turned on in most cells (not just those expressing the GPCR). In one sentence suggest a simple explanation for why cAMP increased in non-GPCR-expressing cells. 9. Taxol is a drug that inhibits microtubule dynamics and is often used for cancer chemotherapy because it preferentially affects dividing cells. What microtubule-based structure is likely disrupted by Taxol so that it specifically affects proliferating cells? 10. Patients with Marfan’s syndrome have a mutation in the fibrillin gene and are at risk of…expand this into 1000 words. the context is Identification of Differentially Expressed Genes in Breast Cancer Using RNA-Seq "In investigating the impact of TONSL overexpression on cellular physiology and gene regulation, differential expression analysis emerges as a pivotal analytical approach. By comparing gene expression profiles between primary cells and TONSL-overexpressing cells, researchers can identify genes whose expression levels are significantly altered. This analysis not only sheds light on the direct targets of TONSL but also unveils broader regulatory networks and pathways influenced by TONSL overexpression. Moreover, by pinpointing key genes involved, researchers can discern potential molecular mechanisms underlying the observed phenotypic changes associated with TONSL overexpression, offering valuable insights into TONSL's role in cellular homeostasis and function. Furthermore, functional enrichment analysis stands as a complementary approach to understanding the…Choose correct option and do explain plz. 1. The protein complex that helps RNA polymerase to cross nucleosomes during extension is:a. SWI-SNFb. poly A polymerasac. TFIISd. FACT 2. The polydenylation is carried out by:a. primaseb. polymers poly ac. a reverse transcriptased. adenylyltransferase 3. The lactose operon produces a polycistronic mRNA that includes four genes: Lacl, LacZ, LacY and LacA. True or false?