RNAP can access the lacz promoter irrespective of whether CAP-CAMP is binding the CAP OTrueO False IlvY is a LysR regulator, and it helps control transcription of a neighboring gene, ilvC. IlvY can eitherbend the DNA and block RNAP's access to ilvC's -35 promoter site, or it can allow access to the ilvC -35site, thereby promoting ilvC transcription. What determines whether IlvY is repressing or activatingtranscription from the ilvC promoter?The presence of the co-inducer acetolactate - acetolactate binds to llvY and changes IlvY's shape such that itdoesn't block the -35 of the ilvC promoter.IlvY represses ilvC transcription when the llvC protein is bound to llvY, and it activates ilvC transcription whenthe llvC protein is not present to bind to llvY.The presence of the co-inducer valine - when valine is present, it binds llvY and causes llvY to represstranscription from the ilvC promoter.The presence of the co-inducer acetolactate - acetolactate induces negative supercoiling at the ilvC promoter,which represses ilvC transcription.

Lac operon is a set of structural genes regulated by a common promoter. It is found in bacteria.

Answered: RNAP can access the lacZ promoter…

Biology: The Unity and Diversity of Life (MindTap Course List)

14th Edition

ISBN:9781305073951

Author:Cecie Starr, Ralph Taggart, Christine Evers, Lisa Starr

Publisher:Cecie Starr, Ralph Taggart, Christine Evers, Lisa Starr

Chapter10: Control Of Gene Expression

Section: Chapter Questions

Problem 2SQ

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A mutation that inactivates the repressor gene of the lac operon results in (a) the continuous transcription of the structural genes (b) no transcription of the structural genes (c) the binding of the repressor to the operator (d) no production of RNA polymerase (e) no difference in the rate of transcription
For the E. coli lac operon, when lactose is present: a. and glucose is absent, cAMP binds and activates catabolicactivator protein (CAP). b. and glucose is absent, the level of cAMP decreases. c. activated CAP binds the repressor protein to remove it from the operator gene. d. the cell prefers lactose over glucose. e. RNA polymerase cannot bind to the promoter.
Binding of _______ to _______ in DNA can increase the rate of transcription of specific genes. a. activators; repressors c. repressors; operators b. activators; enhancers d. repressors; enhancers
Transcriptional regulators are proteins that bind to promoters (the 5-flanking regions of genes) to regulate their transcription. Assume that a particular transcription regulator normally promotes transcription of gene X, a transport protein. If a mutation makes this regulator gene nonfunctional, would the resulting phenotype be similar to a mutation in gene X itself? Why or why not?
Discuss Concepts In a mutant strain of E. coli, the CAP protein is unable to combine with its target region of the lac operon. How would you expect the mutation to affect transcription when cells of this strain are subjected to the following conditions? Lactose and glucose are both available. Lactose is available but glucose is not. Both lactose and glucose are unavailable.
Describe how RBPs can prevent miRNAs from degrading an RNA molecule.
The Events in Transcription Initiation Describe the sequence of events involved in the initiation of transcription by E. coil RNA polymerase. Include in your description those features a gene must have for proper recognition and transcription by RNA poIymerase.
Mechanisms that govern gene expression do not operate during _______. a. transcription c. translation b. RNA processing d. knockouts
Through alternative splicing, eukaryotes (a) reinforce gene inactivation (b) prevent transcription of heterochromatin (c) produce related but different proteins in different tissues (d) amplify genes to meet the requirement of high levels of a gene product (e) bind transcription factors to enhancers to activate transcription
Comparison of Prokaryotic and Eukaryotic Transcription Make a list of the ways that transcription in eukaryotes differs from transcription in prokaryotes.
Quorum sensing is triggered to begin when treatment with antibiotics occurs bacteria release growth hormones bacterial protein expression is switched on a sufficient number of bacteria are present
Suppose that in the formation of phenylalanine hydroxylase mRNA, the exons of the pre-mRNA fail to splice together properly and the resulting enzyme is nonfunctional. This produces an accumulation of high levels of phenylalanine and other compounds, which causes neurological damage. What phenotype would be produced in the affected individual?

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RNAP can access the lacZ promoter irrespective of whether CAP-CAMP is binding the CAP o True False