HYPERVENTILATION DURING KETOACIDOSIS
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WHAT IS THE BIOCHEMISTRY BEHIND THE HYPERVENTILATION DURING KETOACIDOSIS? PLEASE PROVIDE AN ELABORATED ANSWER!
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- Briefly describe the role that phosphofructokinase plays in cellular respiration. Describe the impact a lack of phosphofructokinase would have on cellular respiration and one sign and/or symptom that may be present in someone who suffers from this disorder.Name: Hendric Nduwuba DNP and Cellular Respiration Summary https://www.youtube.com/watch?v=eLtRan2qFx0 Your assignment today is to summarize in two paragraphs (8 sentences total) the effects of dinitrophenol (DNP) on the human body. Make sure you explain what DNP is, the stages of cellular respiration, and how DNP alters the metabolism of the person using it (side effects). The following vocabulary terms need to be used: cellular respiration, adenosine triphosphate, and dinitrophenol (DNP). Your objective is to describe the role of ATP in cellular respiration in order to explain how DNP affects the rate of metabolism. The first paragraph is an introduction to the summary and the second paragraph is an explanation of the main idea. You will be graded on this criteria: main idea, explanation of cellular respiration, explanation of DNP, and paragraph structure.This is a Biochemistry Question! Please select all of the following processes that would INCREASE during untreated or badly managed diabetic ketoacidosis: (More than 1 answer) 1) there is fluid and electrolyte loss in urine 2) plasma pH 3) Glucose uptake & skeletal muscle using as primary fuel 4) Bicarbonate reabsorption & renal Hydrogen excretion 5) Relying on fatty acids & proteins as primary fuels throughout the body 6) Gluconeogenesis, Fatty acid oxidation, & ketogenesis (all within the liver) 7) Lipolysis & fatty acid release from adipose
- Write some alternative names of HMG-CoA? Please answer at your own words.Anemia is characterized by low hemoglobin levels or a reduced number of red blood cells (both of which transport oxygen to cells). A common symptom of anemia is tiredness. Please do not copy answers from another website/source. a) Briefly explain what the consequences are of anemia on cellular energy production. b) Suggest at least one other cellular effect of anemia and a brief explanation of this effect.The level of ketones should be low or absent in MCAD patients?
- Answer the following questions: 1. A patient suffering from fatigue and pallor has been diagnosed of megaloblastic anemia and was given vitamin B12 (cobalamin) as a supplement. Megaloblastic anemia occurs due to reduced activity of methionine synthase, an enzyme responsible for the conversion of homocysteine to methionine. What role is played by vitamin B12 (cobalamin) to methionine synthase? A. apoenzyme B. coenzyme C. holoenzyme D. isoenzymes E. cofactor 2. A given substrate may be acted upon by a number of different enzymes, each of which uses the same substrate(s) and produces the same product(s). The individual numbers of a set of enzymes sharing such characteristics are known as ___, which are of clinical interest because they can be used as molecular markers of tissue damage. A. Cofactor B. Coenzyme C. Holoenzyme D. Apoenzyme E. Isoenzymes 3. Succinic acid dehydrogenase is an enzyme which catalyzes the oxidation of succinic acid during cell respiration. If malonic acid is…Now answer the following questions: 1. Is this patient experiencing a disorder affecting anaerobic or aerobic metabolism? a) disorder affecting anaerobic metabolism. b) disorder affecting aerobic metabolism. c) None of them d) Both 2. You decide to perform assays to check the activity of one or more metabolic enzymes in the red blood cells. Which enzyme(s) would you check? a) Amylase b) Enzymes of pentose phosphate pathways c) Transaminase enzymes d) hexokinase, phosphofructokinase, and pyruvate kinasePlease complete the following question. 5.1
- Write a one-sentence answer to each question. (a) Why does liver damage cause jaundice? (b) Why do individuals with congenital erythropoietic porphyria become anemic? (c) Why does a genetic deficiency of 5,10-methylenetetrahydrofolate reductase cause homocystinuria?what are the metabolic and physiologic capabilities of proteus Vulgaris? describe the different types of metabolism assessed for by physiology tests for proteus Vulgaris.Can you please assist me with these question