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atelets platelet plug formation hemophilia megakaryocytes coagulation thromboxane A2 serotonin agulation fibrin vascular spasm clot Imonary embolism negakaryocytes are huge cells found in red bone marrow that extend long tendrils into blood sinusoids. The flow of blood breaks off small portions of ose tendrils creating the formed element known as platelets atelets are involved in the process of hemostasis. If a blood vessel is severed in an injury, a reflex in the smooth muscle of the vessel wall constricts the broken ssel. This first phase of hemostasis is called vascular spasm This results in reduced blood flow and promotes the success of the remaining two ases of hemostasis. e second phase of hemostasis is platelet plug formation ,where platelets stick to the collagen revealed in the damaged area of the vessel wall. This racts other platelets in a positive feedback mechanism. During this process, platelets degranulate, and release the vasoconstrictor serotonin nd thromboxane A2 , which serves to promote platelet aggregation and vasoconstriction. e final phase of hemostasis is coagulation , where fibrinogen is activated forming fibrin fibrils, that stabilize the platelet ug, producing a Clot emophilia is a clotting disorder, often caused by an inherited clotting factor mutation, where blood fails to clot within a reasonable period of time. is often caused by thrombus forming in a leg vein, breaking free, and lodging in a vessel within the pulmonary circulation.

atelets platelet plug formation hemophilia megakaryocytes coagulation thromboxane A2 serotonin agulation fibrin vascular spasm clot Imonary embolism negakaryocytes are huge cells found in red bone marrow that extend long tendrils into blood sinusoids. The flow of blood breaks off small portions of ose tendrils creating the formed element known as platelets atelets are involved in the process of hemostasis. If a blood vessel is severed in an injury, a reflex in the smooth muscle of the vessel wall constricts the broken ssel. This first phase of hemostasis is called vascular spasm This results in reduced blood flow and promotes the success of the remaining two ases of hemostasis. e second phase of hemostasis is platelet plug formation ,where platelets stick to the collagen revealed in the damaged area of the vessel wall. This racts other platelets in a positive feedback mechanism. During this process, platelets degranulate, and release the vasoconstrictor serotonin nd thromboxane A2 , which serves to promote platelet aggregation and vasoconstriction. e final phase of hemostasis is coagulation , where fibrinogen is activated forming fibrin fibrils, that stabilize the platelet ug, producing a Clot emophilia is a clotting disorder, often caused by an inherited clotting factor mutation, where blood fails to clot within a reasonable period of time. is often caused by thrombus forming in a leg vein, breaking free, and lodging in a vessel within the pulmonary circulation.

Human Anatomy & Physiology (11th Edition)
Human Anatomy & Physiology (11th Edition)
11th Edition
ISBN:9780134580999
Author:Elaine N. Marieb, Katja N. Hoehn
Publisher:Elaine N. Marieb, Katja N. Hoehn
Chapter1: The Human Body: An Orientation
Section: Chapter Questions
Problem 1RQ: The correct sequence of levels forming the structural hierarchy is A. (a) organ, organ system,...
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platelets serotonin platelet plug formation megakaryocytes coagulation coagulation fibrin hemophilia vascular spasm clot thromboxane A2 pulmonary embolism megakaryocytes are huge cells found in red bone marrow that extend long tendrils into blood sinusoids. The flow of blood breaks off small portions of those tendrils creating the formed element known as platelets Platelets are involved in the process of hemostasis. If a blood vessel is severed in an injury, a reflex in the smooth muscle of the vessel wall constricts the broken vessel. This first phase of hemostasis is called vascular spasm This results in reduced blood flow and promotes the success of the remaining two phases of hemostasis. The second phase of hemostasis is platelet plug formation where platelets stick to the collagen revealed in the damaged area of the vessel wall. This attracts other platelets in a positive feedback mechanism. During this process, platelets degranulate, and release the vasoconstrictor serotonin and thromboxane A2 , which serves to promote platelet aggregation and vasoconstriction. The final phase of hemostasis is coagulation where fibrinogen is activated forming fibrin fibrils, that stabilize the platelet plug, producing a Clot hemophilia is a clotting disorder, often caused by an inherited clotting factor mutation, where blood fails to clot within a reasonable period of time. is often caused by thrombus forming in a leg vein, breaking free, and lodging in a vessel within the pulmonary circulation. A
Transcribed Image Text:platelets serotonin platelet plug formation megakaryocytes coagulation coagulation fibrin hemophilia vascular spasm clot thromboxane A2 pulmonary embolism megakaryocytes are huge cells found in red bone marrow that extend long tendrils into blood sinusoids. The flow of blood breaks off small portions of those tendrils creating the formed element known as platelets Platelets are involved in the process of hemostasis. If a blood vessel is severed in an injury, a reflex in the smooth muscle of the vessel wall constricts the broken vessel. This first phase of hemostasis is called vascular spasm This results in reduced blood flow and promotes the success of the remaining two phases of hemostasis. The second phase of hemostasis is platelet plug formation where platelets stick to the collagen revealed in the damaged area of the vessel wall. This attracts other platelets in a positive feedback mechanism. During this process, platelets degranulate, and release the vasoconstrictor serotonin and thromboxane A2 , which serves to promote platelet aggregation and vasoconstriction. The final phase of hemostasis is coagulation where fibrinogen is activated forming fibrin fibrils, that stabilize the platelet plug, producing a Clot hemophilia is a clotting disorder, often caused by an inherited clotting factor mutation, where blood fails to clot within a reasonable period of time. is often caused by thrombus forming in a leg vein, breaking free, and lodging in a vessel within the pulmonary circulation. A
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