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Assume: X-axis is time (20mm or 20 small boxes/second) Y-axis is voltage (10 mm or 10 small boxes/1 mVolt) 21 15 22 25 QUE 1) How much time did it take to complete each cycle of contraction? (RR- Interval) RR #1: RR#2: Is this a normal heart rate?_ RR#3: Average_ Is this sinus bradycardia, normal sinus rhythm, or sinus tachycardia? QUE 2) How much time was required to pass through the AV Node (and atria)? (PR-Interval) PR-I #1: PR-I#2: _PR-I#3: _ Average= Would this time increase or decrease if the person developed a 1st degree heart block later in the day? QUE 3) How much time did the ventricle spend either depolarized or repolarizing? (QT-Interval) QT-I#1: _QT-I#2:_ _QT-I#3: _ Average= If the QT-interval became longer, would the hearts oxygen demands go up or down? QUE 4) How much time did the heart spend generating force? (ST-Segment) What are actin and myosin in the cardiomyocytes doing once calcium has allowed troponin to be removed from myosin? (Assume adequate ATP supplies) QUE 5) How much time did the heart spend in diastole? (time from end of T-wave to start of next P-wave) TP#1: _ TP#2: _ TP#3: Average= If the heart rate increased and the QT-interval was unchanged would the T-P time increase or decrease? WHY? This is tricky, but helps explain why eventually an increasing heart rate means blood and oxygen supply to the heart actually DECREASE at the same time the oxygen demand INCREASES.--> This is a frequent cause of exercise induced angina (localized cardiac hypoxia/ischemia or heart attack).

Assume: X-axis is time (20mm or 20 small boxes/second) Y-axis is voltage (10 mm or 10 small boxes/1 mVolt) 21 15 22 25 QUE 1) How much time did it take to complete each cycle of contraction? (RR- Interval) RR #1: RR#2: Is this a normal heart rate?_ RR#3: Average_ Is this sinus bradycardia, normal sinus rhythm, or sinus tachycardia? QUE 2) How much time was required to pass through the AV Node (and atria)? (PR-Interval) PR-I #1: PR-I#2: _PR-I#3: _ Average= Would this time increase or decrease if the person developed a 1st degree heart block later in the day? QUE 3) How much time did the ventricle spend either depolarized or repolarizing? (QT-Interval) QT-I#1: _QT-I#2:_ _QT-I#3: _ Average= If the QT-interval became longer, would the hearts oxygen demands go up or down? QUE 4) How much time did the heart spend generating force? (ST-Segment) What are actin and myosin in the cardiomyocytes doing once calcium has allowed troponin to be removed from myosin? (Assume adequate ATP supplies) QUE 5) How much time did the heart spend in diastole? (time from end of T-wave to start of next P-wave) TP#1: _ TP#2: _ TP#3: Average= If the heart rate increased and the QT-interval was unchanged would the T-P time increase or decrease? WHY? This is tricky, but helps explain why eventually an increasing heart rate means blood and oxygen supply to the heart actually DECREASE at the same time the oxygen demand INCREASES.--> This is a frequent cause of exercise induced angina (localized cardiac hypoxia/ischemia or heart attack).

Human Anatomy & Physiology (11th Edition)
Human Anatomy & Physiology (11th Edition)
11th Edition
ISBN:9780134580999
Author:Elaine N. Marieb, Katja N. Hoehn
Publisher:Elaine N. Marieb, Katja N. Hoehn
Chapter1: The Human Body: An Orientation
Section: Chapter Questions
Problem 1RQ: The correct sequence of levels forming the structural hierarchy is A. (a) organ, organ system,...
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Assume: X-axis is time (20mm or 20 small boxes/second) Y-axis is voltage (10 mm or 10 small boxes/1 mVolt) 21 15 22 25 QUE 1) How much time did it take to complete each cycle of contraction? (RR- Interval) RR #1: RR#2: Is this a normal heart rate?_ RR#3: Average_ Is this sinus bradycardia, normal sinus rhythm, or sinus tachycardia? QUE 2) How much time was required to pass through the AV Node (and atria)? (PR-Interval) PR-I #1: PR-I#2: _PR-I#3: _ Average= Would this time increase or decrease if the person developed a 1st degree heart block later in the day? QUE 3) How much time did the ventricle spend either depolarized or repolarizing? (QT-Interval) QT-I#1: _QT-I#2:_ _QT-I#3: _ Average= If the QT-interval became longer, would the hearts oxygen demands go up or down? QUE 4) How much time did the heart spend generating force? (ST-Segment) What are actin and myosin in the cardiomyocytes doing once calcium has allowed troponin to be removed from myosin? (Assume adequate ATP supplies) QUE 5) How much time did the heart spend in diastole? (time from end of T-wave to start of next P-wave) TP#1: _ TP#2: _ TP#3: Average= If the heart rate increased and the QT-interval was unchanged would the T-P time increase or decrease? WHY? This is tricky, but helps explain why eventually an increasing heart rate means blood and oxygen supply to the heart actually DECREASE at the same time the oxygen demand INCREASES.--> This is a frequent cause of exercise induced angina (localized cardiac hypoxia/ischemia or heart attack).
Transcribed Image Text:Assume: X-axis is time (20mm or 20 small boxes/second) Y-axis is voltage (10 mm or 10 small boxes/1 mVolt) 21 15 22 25 QUE 1) How much time did it take to complete each cycle of contraction? (RR- Interval) RR #1: RR#2: Is this a normal heart rate?_ RR#3: Average_ Is this sinus bradycardia, normal sinus rhythm, or sinus tachycardia? QUE 2) How much time was required to pass through the AV Node (and atria)? (PR-Interval) PR-I #1: PR-I#2: _PR-I#3: _ Average= Would this time increase or decrease if the person developed a 1st degree heart block later in the day? QUE 3) How much time did the ventricle spend either depolarized or repolarizing? (QT-Interval) QT-I#1: _QT-I#2:_ _QT-I#3: _ Average= If the QT-interval became longer, would the hearts oxygen demands go up or down? QUE 4) How much time did the heart spend generating force? (ST-Segment) What are actin and myosin in the cardiomyocytes doing once calcium has allowed troponin to be removed from myosin? (Assume adequate ATP supplies) QUE 5) How much time did the heart spend in diastole? (time from end of T-wave to start of next P-wave) TP#1: _ TP#2: _ TP#3: Average= If the heart rate increased and the QT-interval was unchanged would the T-P time increase or decrease? WHY? This is tricky, but helps explain why eventually an increasing heart rate means blood and oxygen supply to the heart actually DECREASE at the same time the oxygen demand INCREASES.--> This is a frequent cause of exercise induced angina (localized cardiac hypoxia/ischemia or heart attack).
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