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- discuss chronic Kidney disease: cause(s) of the disorderrisk factors of the disordersigns and/or symptomshow is it diagnosedhow it affects the body overalltreatment(s) and/or cure.what is the prognosis?Which equation should be used to estimate GFR when staging CKD? Which lab value is used to stage severity of AKI? What do you need to look for in a patient's laboratory records to see if they have acute-on-chronic renal injury?The Golden standard for evaluating GFR and renal clearance is urea creatinine creatinine and urea inulin, continuous infusion inulin, single bolus A deficiency of the xanthine oxidase due to severe hepatocellular disease will lead to: increased urea concentration decreased creatinine concentration hyperuricemia hypouricemia xanthinuria None of the above Both C and E Both D and E
- Risk factors for chronic kidney disease identify the dietary and lifestyle components to treat and prevent chronic kidney diseases conditions list food that encourages and discourages for chronic kidney diseases identifypotential medications for chronic kidney disease apply diet management to the treatment of chronicBriefly discuss the drug development of diuretics drugs? Please discuss at your own words(41) A 59 year old woman comes to the physician because of a 3 day history of decreased urine output . She underwent cadaveric renal transplantation. 10 weeks ago, she has hypertension and type-2 diabetes mellitus. She takes immunosuppressive medications including azathioprine, prednisone and tacrolimus.. Physical examination shows tenderness over the allograft. Serum studies show increased urine nitrogen and increased creatinine concentrations. This patients condition is produced by a specific effector cell that targets the transplanted tissue for destruction through the recognition of foreign MHC class 1 antigens. This effector cell most likely expresses and produces which of the following ? Effector Cell Expression. Effector Cell Production (A) CD3 and CD4. Interleukin 2(IL-2) and interferon gamma (B) CD3 and CD 4. IL-4, IL-5 and IL-10 (C) CD3 and CD 8.…
- See attached test results. Mine are marked as N.K Are there any abnormal test results in your urinalysis? If, so indicate any possible causes. If results were normal, can you name any healthy practices that you follow which may have contributed to these results?Pt is a 55 y.o female with past medical history of end-stage renal disease on hemodialysis via perm catheter, hypertension, hyperlipidemia, type 2 diabetes, anemia of chronic disease, peripheral neuropathy, recurrent C.Difficile. History of Acinetobacter bacteremia come into the hospital as direct transfer from Newport given worsening pleural effusion of importance. Pt was recently admitted at Rhode Island Hospital and discharged a few weeks ago after being diagnosed with necrotizing pneumonia. lung disease abscess on CT scan. She underwent BAL and culture grew klebsiella oxytocin which she was treated with Augmentin for 6weeks duration. She had elevated 1,3 Beta D flu an but was deemed to be potentially false positive. She was also found to have Acinetobacter growing from dialysis catheter too and the catheter was removed on 3/8 and a new one was replaced on 3/9. Unfortunately came back from Newport hospital because of progressively worsening shortness of breath as well as well as…Please Answer the problem. There is more to the process of urine formation that meets the eye. Urine by itself is a constantly changing medium by which the needs of the body are automatically addressed by kidneys that work 24/7. Central to this function is the structural and functional unit of the kidney which is the nephron. The failing kidney is the result of nephrons that have failed or are failing to do their function. What can be done? What are the treatment modalities available to a patient in chronic renal failure? What are the considerations and how do these modalities work?
- Please Briefly discuss the drug development of diuretic drugs? Discuss at your own words??.lease answer the following questions: Another patient has signs of glomerular capsule damage (damage to the filtration membrane) and now tests positive for proteins in her urine. What effect, if any, might this have on NFP and why? The same patient is showing signs of edema in her hands and feet (peripheral edema). What might be the explanation for this? What effect might the presence of a kidney stone have on NFP? Explain why?EQ#18 Which letter on the diagram indicates: 8. adrenal gland 9. bladder 10. kidney b a 11. ureter 12. urethra 13. renal artery C 14. renal vein d. f g rowseandembed/index/media-redirect/entryid/1_sdysv1pi/showDescription/false/showTitle/false/showTags/false/showDuration/false/showOwner/false/showUploadDate/false/play MacBook Air
