![Genetics: A Conceptual Approach](https://www.bartleby.com/isbn_cover_images/9781319050962/9781319050962_largeCoverImage.gif)
a.
To explain:
The number of chromosomes in individual with allotriploidy including species I and II.
Introduction:
The chromosomal mutations not only involve the change in the structure of chromosome rather it also includes a change in the number of chromosomes. Aneuploidy is the gain or deletion of one ormore individual chromosome. Polyploidy refers to a gain of the whole set of chromosomes. Polyploidy is of two types, namely- autopolyploidy and allopolyploidy.
a.
![Check Mark](/static/check-mark.png)
Explanation of Solution
Allotriploidy arises when a diploid gamete fuses with haploid gamete. There are two possibilities through which allotriploid can form. One possibility is that it inherits
The number of chromosomes in allotriploid individual including species I and species II if it inherits
Another possibility is that it inherits
The number of chromosomes in allotriploid individual if it inherits
b.
To explain:
The number of chromosomes in individual with autotetraploidy in species II.
Introduction:
The chromosomal mutations not only involve the change in the structure of chromosome rather it also includes a change in the number of chromosomes. Aneuploidy is the gain or deletion of one orthe more individual chromosome. Polyploidy refers to a gain of the whole set of chromosomes. Polyploidy is of two types, namely- autopolyploidy and allopolyploidy.
b.
![Check Mark](/static/check-mark.png)
Explanation of Solution
Autotetraploidy results when there occurs four copies of the whole set of chromosomes in individuals. The autotetraploid individual in species II will have a following number of chromosomes.
The number of chromosomes in autotetraploidindividual of species II will be
c.
To explain:
The number of chromosomes in an individual with trisomy in species I.
Introduction:
The chromosomal mutations not only involve the change in the structure of chromosome rather it also includes a change in the number of chromosomes. Aneuploidy is the gain or deletion of one ormore individual chromosome. Polyploidy refers to a gain of the whole set of chromosomes. Polyploidy is of two types, namely- autopolyploidy and allopolyploidy.
c.
![Check Mark](/static/check-mark.png)
Explanation of Solution
Trisomy is the addition of a single chromosome in the whole set of chromosomes. Addition of single chromosome refers to the gain of one extra homologous copy of individual chromosome. Trisomy is represented as
The number of chromosomes in trisomicindividual of species I will be
d.
To explain:
The number of chromosome in individual with monosomy in species II.
Introduction:
The chromosomal mutations not only involve the change in the structure of chromosome rather it also includes change in the number of chromosomes. Aneuploidy is the gain or deletion of one ofmore individual chromosome. Polyploidy refers to gain of whole set of chromosomes. Polyploidy is of two types, namely- autopolyploidy and allopolyploidy.
d.
![Check Mark](/static/check-mark.png)
Explanation of Solution
Monosomy is the loss of one of the copies of the homologous chromosome. It is represented as
The number of chromosome in individual with monosomy in species II will be
e.
To explain:
The number of chromosome in individual with tetrasomy in species I.
Introduction:
The chromosomal mutations not only involve the change in the structure of chromosome rather it also includes change in the number of chromosomes. Aneuploidy is the gain or deletion of one ofmore individual chromosome. Polyploidy refers to gain of whole set of chromosomes. Polyploidy is of two types, namely- autopolyploidy and allopolyploidy.
e.
![Check Mark](/static/check-mark.png)
Explanation of Solution
Tetrasomy is the acquiring of two nonhomologous chromosomes. It is represented as
The number of chromosome in individual with tetrasomy in species I will be
f.
To explain:
The number of chromosome in individual with allotetraploidy including species I and species II.
Introduction:
The chromosomal mutations not only involve the change in the structure of chromosome rather it also includes change in the number of chromosomes. Aneuploidy is the gain or deletion of one ofmore individual chromosome. Polyploidy refers to gain of whole set of chromosomes. Polyploidy is of two types, namely- autopolyploidy and allopolyploidy.
f.
![Check Mark](/static/check-mark.png)
Explanation of Solution
Allotetraploidy arises when a diploid gamete fuses with diploid gamete or a haploid gamete fuses with triploid gamete. The number of chromosomes in allotetraploid if it inherits
There are two possibilities through which allotetraploid can form. The first possibility is that it inherits
The number of chromosomes in allotetraploid individual if it inherits
The second possibility is that it inherits
The number of chromosomes in allotetraploid individual if it inherits
Conclusion:
Autopolyploids are produced because of mistakes which might occur during mitosis or meiosis which results in the production of extra sets of chromosomes. Aneuploidy can be defined as the increase or decrease in the individual chromosome number. Aneuploidy might arise because of the loss of chromosome during mitosis or meiosis or nondisjunction of homologous chromosomes or sister chromatids. There are four types of aneuploidy, namely- nullisomy, monosomy, trisomy, and tetrasomy.
Want to see more full solutions like this?
Chapter 8 Solutions
Genetics: A Conceptual Approach
- What are some external influences that keep people from making healthy eating decisions?arrow_forwardWhat type of structure(s) would you expect to see in peripheral membrane proteins? (mark all that apply) A. Amphipathic alpha helix (one side is hydrophilic and one side is hydrophobic) B. A hydrophobic beta barrel C. A hydrophobic alpha helix D. A chemical group attached to the protein that can anchor it to the membranearrow_forwardTemporal flexibility (the ability to change over time) of actin structures within a cell is maintained by… A. The growth/shrinkage cycle B. Periodic catastrophe C. GTP hydrolysis D. Treadmilling E. None of the abovearrow_forward
- During in vitro polymerization of actin and microtubule filaments from their subunits, what causes the initial delay in filament growth? A.Nucleation B.Reaching homeostasis C.Nucleotide exchange D.ATP or GTP hydrolysis E.Treadmillingarrow_forwardYou expect to find which of the following in the Microtubule Organizing Center (MTOC)...(mark all that apply) A. Gamma tubulin B. XMAP215 C. Centrioles D. Kinesin-13arrow_forwardThe actin-nucleating protein formin has flexible “arms” containing binding sites that help recruit subunits in order to enhance microfilament polymerization. What protein binds these sites? A.Thymosin B.Profilin C.Cofilin D.Actin E.Tropomodulinarrow_forward
- While investigating an unidentified motor protein, you discover that it has two heads that bind to actin. Based on this information, you could confidently determine that it is NOT... (mark all that apply) A. A myosin I motor B. A dynein motor C. A myosin VI motor D. A kinesin motorarrow_forwardYou isolate the plasma membrane of cells and find that . . . A. it contains regions with different lipid compositions B. it has different lipid types on the outer and cytosolic leaflets of the membrane C. neither are possible D. A and B both occurarrow_forwardYou are studying the mobility of a transmembrane protein that contains extracellular domains, one transmembrane domain, and a large cytosolic domain. Under normal conditions, this protein is confined to a particular region of the membrane due to the cortical actin cytoskeletal network. Which of the following changes is most likely to increase mobility of this protein beyond the normal restricted region of the membrane? A. Increased temperature B. Protease cleavage of the extracellular domain of the protein C. Binding to a free-floating extracellular ligand, such as a hormone D. Protease cleavage of the cytosolic domain of the protein E. Aggregation of the protein with other transmembrane proteinsarrow_forward
- Topic: Benthic invertebrates as an indicator species for climate change, mapping changes in ecosystems (Historical Analysis & GIS) What objects or events has the team chosen to analyze? How does your team wish to delineate the domain or scale in which these objects or events operate? How does that limited domain facilitate a more feasible research project? What is your understanding of their relationships to other objects and events? Are you excluding other things from consideration which may influence the phenomena you seek to understand? Examples of such exclusions might include certain air-born pollutants; a general class of water bodies near Ottawa, or measurements recorded at other months of the year; interview participants from other organizations that are involved in the development of your central topic or issue. In what ways do your research questions follow as the most appropriate and/or most practical questions (given the circumstances) to pursue to better understand…arrow_forwardThe Esp gene encodes a protein that alters the structure of the insulin receptor on osteoblasts and interferes with the binding of insulin to the receptor. A researcher created a group of osteoblasts with an Esp mutation that prevented the production of a functional Esp product (mutant). The researcher then exposed the mutant strain and a normal strain that expresses Esp to glucose and compared the levels of insulin in the blood near the osteoblasts (Figure 2). Which of the following claims is most consistent with the data shown in Figure 2 ? A Esp expression is necessary to prevent the overproduction of insulin. B Esp protein does not regulate blood-sarrow_forwardPredict the per capita rate of change (r) for a population of ruil trees in the presence of the novel symbiont when the soil moisture is 29%. The formula I am given is y= -0.00012x^2 + 0.0088x -0.1372. Do I use this formula and plug in 29 for each x variable?arrow_forward
- Human Anatomy & Physiology (11th Edition)BiologyISBN:9780134580999Author:Elaine N. Marieb, Katja N. HoehnPublisher:PEARSONBiology 2eBiologyISBN:9781947172517Author:Matthew Douglas, Jung Choi, Mary Ann ClarkPublisher:OpenStaxAnatomy & PhysiologyBiologyISBN:9781259398629Author:McKinley, Michael P., O'loughlin, Valerie Dean, Bidle, Theresa StouterPublisher:Mcgraw Hill Education,
- Molecular Biology of the Cell (Sixth Edition)BiologyISBN:9780815344322Author:Bruce Alberts, Alexander D. Johnson, Julian Lewis, David Morgan, Martin Raff, Keith Roberts, Peter WalterPublisher:W. W. Norton & CompanyLaboratory Manual For Human Anatomy & PhysiologyBiologyISBN:9781260159363Author:Martin, Terry R., Prentice-craver, CynthiaPublisher:McGraw-Hill Publishing Co.Inquiry Into Life (16th Edition)BiologyISBN:9781260231700Author:Sylvia S. Mader, Michael WindelspechtPublisher:McGraw Hill Education
![Text book image](https://www.bartleby.com/isbn_cover_images/9780134580999/9780134580999_smallCoverImage.gif)
![Text book image](https://www.bartleby.com/isbn_cover_images/9781947172517/9781947172517_coverImage_Textbooks.gif)
![Text book image](https://www.bartleby.com/isbn_cover_images/9781259398629/9781259398629_smallCoverImage.gif)
![Text book image](https://www.bartleby.com/isbn_cover_images/9780815344322/9780815344322_smallCoverImage.gif)
![Text book image](https://www.bartleby.com/isbn_cover_images/9781260159363/9781260159363_smallCoverImage.gif)
![Text book image](https://www.bartleby.com/isbn_cover_images/9781260231700/9781260231700_smallCoverImage.gif)