Bundle: Biology: The Unity and Diversity of Life, Loose-leaf Version, 14th + LMS Integrated for MindTap Biology, 2 terms (12 months) Printed Access Card
14th Edition
ISBN: 9781305775480
Author: Cecie Starr, Ralph Taggart, Christine Evers, Lisa Starr
Publisher: Cengage Learning
expand_more
expand_more
format_list_bulleted
Question
Chapter 42, Problem 3CT
Summary Introduction
To explain: Tumors from germ cells contain any type of tissue, whereas the tumors from somatic cells have only limited number of tissues.
Concept introduction: Uncontrolled growth of cells due to the defects in the cell cycle results in tumors. Tumors can be benign or malignant. The gastrula has three germ layers. The germ layers undergo organogenesis to form the organ systems, and finally form the complete organism.
Expert Solution & Answer
Want to see the full answer?
Check out a sample textbook solutionStudents have asked these similar questions
What is the difference between benign and malignant tumors? What is the disadvantage of this classification?
Which is true for cancer cells:
1) Cell death occurs after a determined number of cell divisions
2) Contact with other cells reduces chance of cell division
3) Cell division occurs in the presence of stop signals.
Explain why metastasis can lead to multiplesecondary tumors in different sites.
Chapter 42 Solutions
Bundle: Biology: The Unity and Diversity of Life, Loose-leaf Version, 14th + LMS Integrated for MindTap Biology, 2 terms (12 months) Printed Access Card
Ch. 42 - The end product of cleavage is a __________. a....Ch. 42 - The outermost germ layer in a vertebrate gastrula...Ch. 42 - Prob. 3SQCh. 42 - Match each term with the most suitable...Ch. 42 - In humans, fertilization typically occurs in the...Ch. 42 - The ________, a fluid-filled sac, surrounds and...Ch. 42 - Prob. 1DAACh. 42 - Prob. 2DAACh. 42 - Prob. 3DAACh. 42 - Birth Defects and Multiple Births A woman who...
Ch. 42 - The placenta consists of __________. a. embryonic...Ch. 42 - During the fetal period, ____________. a....Ch. 42 - Prob. 9SQCh. 42 - Match each hormone with its action(s). ______...Ch. 42 - Prob. 11SQCh. 42 - Prob. 12SQCh. 42 - Prob. 13SQCh. 42 - Prob. 14SQCh. 42 - Prob. 15SQCh. 42 - The rubella virus causes German measles. If a...Ch. 42 - Prob. 2CTCh. 42 - Prob. 3CT
Knowledge Booster
Learn more about
Need a deep-dive on the concept behind this application? Look no further. Learn more about this topic, biology and related others by exploring similar questions and additional content below.Similar questions
- Explain why mutations in tumor suppressor genes are recessive (both copies of the gene must be defective for the regulation of cell division to be defective), whereas mutations in oncogenes are dominant.arrow_forwardTissues and differentiation a)Explain what is meant by termination and differentiation ).b) Explain the difference between an oncogenic and a tumour suppressor gene and describe how they are involved in the onset of cancerarrow_forwardTissues and differentiation: a) Explain what is meant by determination and differentiation b) Explain the difference between an oncogene and a tumor suppressor gene and describe how they are involved in the development of cancerarrow_forward
- Although cancer is not a contagious disease in humans or other vertebrates, there have been rare cases in which cancers have spread from one organism to another. Describe three cases of these contagious cancers and what conditions might have led to their appearance. For an introduction to this topic, see http:// www.cancer.org/cancer/cancerbasics/is-cancer-contagious.arrow_forwardWhen some cancer cells produce growth factors that stimulate their own division leading to continuous self- stimulation for cell division it is referred to as a) TNF tumor necrosis factor b) AGS autocrine growth stimulationarrow_forwardExcessive angiogenesis and reduced apoptosis are two of the six characteristics that define cancerous tumors. Discuss how developing treatments for these two conditions might prevent the development of cancer altogether from pre-malignant tumors.arrow_forward
- #1) Hyperplastic growth are not cancerous yet but show a higher than normal proliferation rate. True False #2) When cancer cells have not spread beyond its original site, the term used to describe it is benign growth intraepithelial neoplasia carcinoma stage 3 carcinoma in situ #9) Cancer cells generally have missense mutations in p53 gene, resulting in truncated p53 normally active p53 dominant negative p53 inactive p53 #10) Single or double stranded breaks in DNA activate Chk 1 and 2 kinases, which phosphorylates p53. This results in --- in the level of p53 in the cell. increase decreasearrow_forwardWhat is the fastest rate of growth of the tumor?arrow_forwardList and define the two general types of tumor.arrow_forward
- Differentiate among the terms cancer, tumor, and neoplasm. Note. There is a little confusion about the terms "tumor" and "neoplasm". Think about cancers such as leukemia and other blood cell cancers. Are they tumors?arrow_forwardDiscuss the complete cell cycle in a human cell, mitosis and meiosis, and the regulatory components (i.e. the proteins associated with cellular checkpoints) of the cell cycle. Tumor growth results when the cell cycle checkpoints are ignored. Give an example of how tumor growth could result from either a loss-of-function or a gain-of-function mutation.arrow_forwardAnswer the following with respect to Caner.(a) How does a cancerous cell differ from a normal cell?(b) Benign tumor is less dangerous than malignant tumor. Why(c) Describe causes of cancer.(d) mention two methods of treatment of the disease.arrow_forward
arrow_back_ios
SEE MORE QUESTIONS
arrow_forward_ios
Recommended textbooks for you
- Human Anatomy & Physiology (11th Edition)BiologyISBN:9780134580999Author:Elaine N. Marieb, Katja N. HoehnPublisher:PEARSONBiology 2eBiologyISBN:9781947172517Author:Matthew Douglas, Jung Choi, Mary Ann ClarkPublisher:OpenStaxAnatomy & PhysiologyBiologyISBN:9781259398629Author:McKinley, Michael P., O'loughlin, Valerie Dean, Bidle, Theresa StouterPublisher:Mcgraw Hill Education,
- Molecular Biology of the Cell (Sixth Edition)BiologyISBN:9780815344322Author:Bruce Alberts, Alexander D. Johnson, Julian Lewis, David Morgan, Martin Raff, Keith Roberts, Peter WalterPublisher:W. W. Norton & CompanyLaboratory Manual For Human Anatomy & PhysiologyBiologyISBN:9781260159363Author:Martin, Terry R., Prentice-craver, CynthiaPublisher:McGraw-Hill Publishing Co.Inquiry Into Life (16th Edition)BiologyISBN:9781260231700Author:Sylvia S. Mader, Michael WindelspechtPublisher:McGraw Hill Education
Human Anatomy & Physiology (11th Edition)
Biology
ISBN:9780134580999
Author:Elaine N. Marieb, Katja N. Hoehn
Publisher:PEARSON
Biology 2e
Biology
ISBN:9781947172517
Author:Matthew Douglas, Jung Choi, Mary Ann Clark
Publisher:OpenStax
Anatomy & Physiology
Biology
ISBN:9781259398629
Author:McKinley, Michael P., O'loughlin, Valerie Dean, Bidle, Theresa Stouter
Publisher:Mcgraw Hill Education,
Molecular Biology of the Cell (Sixth Edition)
Biology
ISBN:9780815344322
Author:Bruce Alberts, Alexander D. Johnson, Julian Lewis, David Morgan, Martin Raff, Keith Roberts, Peter Walter
Publisher:W. W. Norton & Company
Laboratory Manual For Human Anatomy & Physiology
Biology
ISBN:9781260159363
Author:Martin, Terry R., Prentice-craver, Cynthia
Publisher:McGraw-Hill Publishing Co.
Inquiry Into Life (16th Edition)
Biology
ISBN:9781260231700
Author:Sylvia S. Mader, Michael Windelspecht
Publisher:McGraw Hill Education
Cell Differentiation | Genetics | Biology | FuseSchool; Author: FuseSchool - Global Education;https://www.youtube.com/watch?v=gwAz_BtVuLA;License: Standard YouTube License, CC-BY