The overstimulation of a muscle fiber is prevented by the role of the enzyme acetylcholinesterase (ACh). The enzyme breaks down excess ACh into inactive molecules in the synapse, and this ends neurotransmission.

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Chapter 35, Problem 7TY

Summary Introduction

Introduction:

The overstimulation of a muscle fiber is prevented by the role of the enzyme acetylcholinesterase (ACh). The enzyme breaks down excess ACh into inactive molecules in the synapse, and this ends neurotransmission.

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Answer 2

Question

Chapter 35, Problem 7TY

Summary Introduction

Introduction:

The overstimulation of a muscle fiber is prevented by the role of the enzyme acetylcholinesterase (ACh). The enzyme breaks down excess ACh into inactive molecules in the synapse, and this ends neurotransmission.

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Answer 3

Question

Chapter 35, Problem 7TY

Summary Introduction

Introduction:

The overstimulation of a muscle fiber is prevented by the role of the enzyme acetylcholinesterase (ACh). The enzyme breaks down excess ACh into inactive molecules in the synapse, and this ends neurotransmission.

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Answer 4

Question

Chapter 35, Problem 7TY

Summary Introduction

Introduction:

The overstimulation of a muscle fiber is prevented by the role of the enzyme acetylcholinesterase (ACh). The enzyme breaks down excess ACh into inactive molecules in the synapse, and this ends neurotransmission.

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Answer 5

Chapter 35, Problem 7TY

Summary Introduction

Introduction:

The overstimulation of a muscle fiber is prevented by the role of the enzyme acetylcholinesterase (ACh). The enzyme breaks down excess ACh into inactive molecules in the synapse, and this ends neurotransmission.

Answer 6

Chapter 35, Problem 7TY

Summary Introduction

Introduction:

The overstimulation of a muscle fiber is prevented by the role of the enzyme acetylcholinesterase (ACh). The enzyme breaks down excess ACh into inactive molecules in the synapse, and this ends neurotransmission.

Answer 7

Chapter 35, Problem 7TY

Summary Introduction

Introduction:

The overstimulation of a muscle fiber is prevented by the role of the enzyme acetylcholinesterase (ACh). The enzyme breaks down excess ACh into inactive molecules in the synapse, and this ends neurotransmission.

Answer 8

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Answer 11

Ch. 35.1 - Prob. 1CC Ch. 35.1 - Prob. 1TYK Ch. 35.1 - Prob. 2TYK Ch. 35.2 - Prob. 1CC Ch. 35.2 - Rigor mortis is the condition that occurs shortly...Ch. 35.2 - Prob. 1BC Ch. 35.2 - Prob. 3CC Ch. 35.2 - Prob. 2BC Ch. 35.2 - Prob. 1TYK Ch. 35.2 - Prob. 2TYK

The overstimulation of a muscle fiber is prevented by the role of the enzyme acetylcholinesterase (ACh). The enzyme breaks down excess ACh into inactive molecules in the synapse, and this ends neurotransmission.

Solution Summary: The author explains that overstimulation of a muscle fiber is prevented by the enzyme acetylcholinesterase (ACh), which breaks down excess ACh into inactive molecules in the synapse

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Introduction:

The overstimulation of a muscle fiber is prevented by the role of the enzyme acetylcholinesterase (ACh). The enzyme breaks down excess ACh into inactive molecules in the synapse, and this ends neurotransmission.

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Chapter 35 Solutions

The overstimulation of a muscle fiber is prevented by the role of the enzyme acetylcholinesterase (ACh). The enzyme breaks down excess ACh into inactive molecules in the synapse, and this ends neurotransmission.

Solution Summary: The author explains that overstimulation of a muscle fiber is prevented by the enzyme acetylcholinesterase (ACh), which breaks down excess ACh into inactive molecules in the synapse

Concept explainers

Videos

Introduction: The overstimulation of a muscle fiber is prevented by the role of the enzyme acetylcholinesterase (ACh). The enzyme breaks down excess ACh into inactive molecules in the synapse, and this ends neurotransmission.

Want to see the full answer?

Chapter 35 Solutions

Introduction:

The overstimulation of a muscle fiber is prevented by the role of the enzyme acetylcholinesterase (ACh). The enzyme breaks down excess ACh into inactive molecules in the synapse, and this ends neurotransmission.