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To determine:
The differences between the pathogenicity of coliform, noncoliform, and truly pathogenic enteric bacteria.
Introduction:
The pathogenicity in Enterobacteria is affected by various factors like: Exotoxins, which leads to symptoms like diarrhea. Fimbriae and adhesion, which help the bacteria to adhere to host cells. A polysaccharide capsule prevents the cell from phagocytosis and antibodies. Iron binding compounds. Type III secretion system, synthesizes polypeptides. Lipooligosaccharides in the outermost membrane of the cell wall, which consists of Lipid A that causes symptoms like fever and dilation of blood vessels. Hemolysin breaks down red blood cells in the host.
Pathogenic members of the family Enterobacteriaceae are categorized into three groups:
Coliforms: These are pathogens which are opportunistic and can ferment lactose at a very fast rate. They are found in the usual microbial habitat. For example: E. coli, Klebsiella, and Citrobacter.
Noncoliforms: These too are pathogens which are quite opportunistic and do not ferment lactose. They are also found in the usual microbial habitat. For example: Proteus, Morganella, and Providencia.
True pathogens: These bacteria do not ferment lactose and don’t live in the usual habitat of microbes. They have many virulent factors which make them always pathogenic. For example: Salmonella, Shigella, and Yesinia.
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Chapter 20 Solutions
Microbiology with Diseases by Taxonomy (5th Edition)
- 18. Watch this short youtube video about SARS CoV-2 replication. SARS-CoV-2 Life Cycle (Summer 2020) - YouTube.19. What is the name of the receptor that SARS CoV-2 uses to enter cells? Which human cells express this receptor? 20. Name a few of the proteins that the SARS CoV-2 mRNA codes for. 21. What is the role of the golgi apparatus related to SARS CoV-2arrow_forwardState the five functions of Globular Proteins, and give an example of a protein for each function.arrow_forwardDiagram of check cell under low power and high powerarrow_forward
- a couple in which the father has the a blood type and the mother has the o blood type produce an offspring with the o blood type, how does this happen? how could two functionally O parents produce an offspring that has the a blood type?arrow_forwardWhat is the opening indicated by the pointer? (leaf x.s.) stomate guard cell lenticel intercellular space none of thesearrow_forwardIdentify the indicated tissue? (stem x.s.) parenchyma collenchyma sclerenchyma ○ xylem ○ phloem none of thesearrow_forward
- Where did this structure originate from? (Salix branch root) epidermis cortex endodermis pericycle vascular cylinderarrow_forwardIdentify the indicated tissue. (Tilia stem x.s.) parenchyma collenchyma sclerenchyma xylem phloem none of thesearrow_forwardIdentify the indicated structure. (Cucurbita stem l.s.) pit lenticel stomate tendril none of thesearrow_forward
- Identify the specific cell? (Zebrina leaf peel) vessel element sieve element companion cell tracheid guard cell subsidiary cell none of thesearrow_forwardWhat type of cells flank the opening on either side? (leaf x.s.) vessel elements sieve elements companion cells tracheids guard cells none of thesearrow_forwardWhat specific cell is indicated. (Cucurbita stem I.s.) vessel element sieve element O companion cell tracheid guard cell none of thesearrow_forward
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