ANATOMY & PHYSIOLOGY 4/E PAC 1 SEMESTER
4th Edition
ISBN: 9781265061173
Author: McKinley
Publisher: MCG CUSTOM
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Textbook Question
Chapter 19.6, Problem 23WDYL
What is autorhythmicity? Describe how nodal cells function as autorhythmic cells to serve as the pacemaker of the heart.
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What is autorhythmicity? Describe how nodal cells function as autorhythmic cells to serve as the pacemaker of the heart.
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Chapter 19 Solutions
ANATOMY & PHYSIOLOGY 4/E PAC 1 SEMESTER
Ch. 19.1 - Prob. 1WDYLCh. 19.1 - Prob. 2WDYLCh. 19.1 - Prob. 3WDYLCh. 19.1 - Prob. 4WDYLCh. 19.1 - Prob. 5WDYLCh. 19.2 - What is the bony structure that protects both the...Ch. 19.2 - Prob. 7WDYLCh. 19.2 - Prob. 8WDYLCh. 19.3 - Prob. 9WDYLCh. 19.3 - Prob. 10WDYL
Ch. 19.3 - Prob. 11WDYLCh. 19.3 - Prob. 12WDYLCh. 19.3 - Prob. 13WDYLCh. 19.3 - Prob. 14WDYLCh. 19.3 - Prob. 15WDYLCh. 19.3 - Prob. 16WDYLCh. 19.3 - What areas of the heart are deprived of blood when...Ch. 19.3 - Prob. 18WDYLCh. 19.4 - Prob. 19WDYLCh. 19.5 - Prob. 20WDYLCh. 19.5 - Which autonomic division is associated with the...Ch. 19.6 - Prob. 22WDYLCh. 19.6 - What is autorhythmicity? Describe how nodal cells...Ch. 19.6 - What is the path of an action potential through...Ch. 19.6 - What anatomic features slow the conduction rate of...Ch. 19.7 - In which direction does Ca2+ move in response to...Ch. 19.7 - What three electrical events occur at the...Ch. 19.7 - What is the significance of the extended...Ch. 19.7 - What events in the heart are indicated by each of...Ch. 19.8 - Pressure changes that occur during the cardiac...Ch. 19.8 - What is occurring during ventricular ejection?Ch. 19.8 - Prob. 32WDYLCh. 19.8 - Define end-diastolic volume, end-systolic volume,...Ch. 19.9 - What are the two factors that determine cardiac...Ch. 19.9 - What is the cardiac output at rest and during...Ch. 19.9 - Prob. 36WDYLCh. 19.9 - Describe the atrial reflex, which involves...Ch. 19.9 - Prob. 38WDYLCh. 19.9 - Prob. 39WDYLCh. 19.10 - What would be the path of blood flow through the...Ch. 19 - Which of the following is the correct circulatory...Ch. 19 - The pericardial cavity is located between the a....Ch. 19 - How is blood prevented from backflowing from the...Ch. 19 - ____ 4. Venous blood draining from the heart wall...Ch. 19 - _____ 5. Calcium channels in the nodal cells...Ch. 19 - ____6. Action potentials are spread rapidly...Ch. 19 - Why is it necessary to stimulate papillary muscles...Ch. 19 - ____ 8. Preload is a measure of a. stretch of...Ch. 19 - ____ 9. All of the following occur when the...Ch. 19 - ____10. What occurs during the atrial reflex? a....Ch. 19 - Prob. 11DYKBCh. 19 - Compare the structure, location, and function of...Ch. 19 - Prob. 13DYKBCh. 19 - Explain why the walls of the atria are thinner...Ch. 19 - Describe the structure and function of...Ch. 19 - Explain the general location and function of...Ch. 19 - Describe the functional differences in the effects...Ch. 19 - Prob. 18DYKBCh. 19 - List the five events of the cardiac cycle, and...Ch. 19 - Define cardiac output, and explain how it is...Ch. 19 - A young man was doing some vigorous exercise when...Ch. 19 - A young man was doing some vigorous exercise when...Ch. 19 - Prob. 3CALCh. 19 - Prob. 4CALCh. 19 - During surgery, the right vagus nerve was...Ch. 19 - Prob. 1CSLCh. 19 - Prob. 2CSLCh. 19 - Your grandfather was told that his SA node...
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- Although not considered the primary pacemaker of the heart, the atrioventricular node can indeed be considered as playing SOME role in setting the rhythm of the heart (kind of a secondary pacemaker). Explain how/why.arrow_forwardDescribe why a doctor would give a patient that is diagnosed with acute myocardial infarction the treatment of tissue plasminogen activator(tPA)? What will it do?arrow_forwardWhat are the difference between contractile cells of the heart and electrical cells responsible for generation and conduction of impulses. Why is the cardiac action potential propagated more slowly in an AV node cell than in an atrial or ventricular myocyte? How are the channels and ions that are responsible for depolarization and repolarization work? How can be AV conduction affected by hypokalemia?arrow_forward
- Describe the functional differences in the effects of the sympathetic and parasympathetic divisions of the autonomic nervous system on the activity of the heart.arrow_forwardWhat is the mechanism behind the action potentials in the pacemaker cells?arrow_forwardDraw these five graphs on the same set of axes: atrial autorhythmic cell action potential atrial cardiac contractile cell action potential ventricular autorhythmic cell action potential ventricular cardiac contractile cell action potential ventricular myocardial muscle tensionarrow_forward
- We analyzed the difference between the action potential of a cardiac contractile cell and an action potential in a neuron. Describe the physiological mechanisms behind the primary difference in the shape of these two action potentials. Why is this feature important to the normal workings of the heart?arrow_forwardA variety of ways to calculate heart rate from an ECG may be used. The quickest way to calculate heart rate on a rhythm strip is: Count the number of large squares between one R-R interval, and divide this number into 1500. Calculate the number of QRS complexes in 1 minute Count the number of R-R intervals in 6 seconds, and multiply by 10 Count the number of small squares between one R-R interval, and multiply this number by 300.arrow_forwardDescribe the pathway of electrical conduction of the heart, starting with the SA node. How does damage to the AV node affect this conduction pathway and the ECG?arrow_forward
- Given the role of potassium in myocardial cell action potentials, explain how hyperkalemia can stop the heart. Make sure to explain in terms of ion movementsarrow_forwardDescribe the mechanical events of the cardiac cycle. (use terms like atrial systole, atrial diastole, ventricular diastole, ventricular systole, isovolumic ventricular contraction, isovolumic ventricular relaxation, ventricular ejection, period of rapid filling)arrow_forwardThe exact cause of blood clotting in the left atrium of the heart during atrial fibrillation (AF) is not known. In part it might be due to blood stasis (not moving) or it may result in combination with endothelial cells damage. (A) What are the potential mechanisms that might drive clotting and what markers might you look for in the blood to predict potential risk? (B) Other than treating the arrhythmia, what other pharmacological intervention might be provided to AF sufferers?arrow_forward
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