Nester's Microbiology: A Human Perspective
9th Edition
ISBN: 9781259709999
Author: Denise G. Anderson Lecturer, Sarah Salm, Deborah Allen
Publisher: McGraw-Hill Education
expand_more
expand_more
format_list_bulleted
Concept explainers
Question
Chapter 19, Problem 12SA
Summary Introduction
To review:
Themain reservoirs of nosocomial infection.
Introduction:
The patient in a healthcare setting is exposed to a wide variety of pathogens. These pathogens are antimicrobial resistant and also virulent. Hence, the patients are more prone to acquire infections while receiving treatment in the hospitals. These infections that are acquired while receiving treatment in hospitals are called Healthcare-associated infections (HAIs).
The hospital-acquired infections are also called nosocomial infections.
Expert Solution & Answer
Want to see the full answer?
Check out a sample textbook solution
Students have asked these similar questions
Stage
Percent
Time in Hours
Interphase
.60
14.4
Prophase
.20
4.8
Metaphase
.10
2.4
Anaphase
.06
1.44
Telophase
.03
.72
Cytukinesis
.01
.24
Can you summarize the results in the chart and explain which phases are faster and why the slower ones are slow?
Can you circle a cell in the different stages of mitosis?
1.prophase
2.metaphase
3.anaphase
4.telophase
5.cytokinesis
Which microbe does not live part of its lifecycle outside humans?
A. Toxoplasma gondii
B. Cytomegalovirus
C. Francisella tularensis
D. Plasmodium falciparum
explain your answer thoroughly.
Chapter 19 Solutions
Nester's Microbiology: A Human Perspective
Ch. 19 - Compare the impact on a society of an endemic...Ch. 19 - What is the epidemiological significance of people...Ch. 19 - Prob. 3SACh. 19 -
4. List the main portals of exit from the human...Ch. 19 -
5. Name the most important control measure for...Ch. 19 - Prob. 6SACh. 19 - Draw representative epi curves (time versus number...Ch. 19 - Differentiate among a case-control study, a...Ch. 19 -
9. What information is available in the Weekly...Ch. 19 - How does the assignment of individuals to groups...
Ch. 19 - Describe the factors that contribute to the...Ch. 19 - Prob. 12SACh. 19 -
1. Which of the following is an example of a...Ch. 19 -
2. Which of the following would be the easiest to...Ch. 19 -
3. Which of the following methods of disease...Ch. 19 - Which of the following statements is false? a) A...Ch. 19 - Which of the following statements is false? a) A...Ch. 19 - Prob. 6MCCh. 19 - Prob. 7MCCh. 19 -
8. All of the following are thought to contribute...Ch. 19 - Prob. 9MCCh. 19 -
10. An endemic disease
a) is typically...Ch. 19 - Prob. 1ACh. 19 - Prob. 2ACh. 19 - Prob. 1CTCh. 19 - As shown in the graphs that follow, the proportion...
Knowledge Booster
Learn more about
Need a deep-dive on the concept behind this application? Look no further. Learn more about this topic, biology and related others by exploring similar questions and additional content below.Similar questions
- Select all of the following that the ablation (knockout) or ectopoic expression (gain of function) of Hox can contribute to. Another set of wings in the fruit fly, duplication of fingernails, ectopic ears in mice, excess feathers in duck/quail chimeras, and homeosis of segment 2 to jaw in Hox2a mutantsarrow_forwardSelect all of the following that changes in the MC1R gene can lead to: Changes in spots/stripes in lizards, changes in coat coloration in mice, ectopic ear formation in Siberian hamsters, and red hair in humansarrow_forwardPleiotropic genes are genes that (blank) Cause a swapping of organs/structures, are the result of duplicated sets of chromosomes, never produce protein products, and have more than one purpose/functionarrow_forward
- A loss of function mutation in Pitx1 enhancers can cause (blank) Removal of Pitx1 exons and growth of ectopic hindlimbs, growth of extra ectopic forelimbs, loss of forelimb specification and development, and loss of hindlimb specification and developmentarrow_forwardHox1a most likely contributes to (blank) patterning in the developing embryo? Ventral, posterior, limb or anteriorarrow_forwardSelect all of the following that can help establish Hox gene expression boundaries (things that affect Hox and not things that Hox affects). Retinoic acid, anterior/posterior axis, fibroblast growth factors, vagal neural crest, and enhancersarrow_forward
- Ectopic expression of Hox often results in (blank) phenotypes. (Blank) transformations are characterized by the replacement of one body part/structure with another. Hoxeotic, homealoneotic, joexotic, or homeoticarrow_forwardWhat's the difference when drawing omega-6 and omega-3?arrow_forward. Consider a base substitution mutation that occurred in a DNA sequence that resulted in a change in the encoded protein from the amino acid glutamic acid to aspartic acid. Normally the glutamic acid amino acid is located on the outside of the soluble protein but not near an active site. O-H¨ A. What type of mutation occurred? O-H B. What 2 types of chemical bonds are found in the R-groups of each amino acid? The R groups are shaded. CH2 CH2 CH2 H2N-C-COOH H2N-C-COOH 1 H Glutamic acid H Aspartic acid C. What 2 types of bonds could each R-group of each of these amino acids form with other molecules? D. Consider the chemical properties of the two amino acids and the location of the amino acid in the protein. Explain what effect this mutation will have on this protein's function and why.arrow_forward
- engineered constructs that consist of hollow fibers are acting as synthetic capillaries, around which cells have been loaded. The cellular space around a single fiber can be modeled as if it were a Krogh tissue cylinder. Each fiber has an outside “capillary” radius of 100 µm and the “tissue” radius can be taken as 200 µm. The following values apply to the device:R0 = 20 µM/secaO2 = 1.35 µM/mmHgDO2,T = 1.67 x 10-5 cm2/secPO2,m = 4 x 10-3 cm/secInstead of blood inside the fibers, the oxygen transport and tissue consumption are being investigated by usingan aqueous solution saturated with pure oxygen. As a result, there is no mass transfer resistance in the synthetic“capillary”, only that due to the membrane itself. Rather than accounting for pO2 variations along the length ofthe fiber, use an average value in the “capillary” of 130 mmHg.Is the tissue fully oxygenated?arrow_forwardMolecular Biology Please help with question. thank you You are studying the expression of the lac operon. You have isolated mutants as described below. In the presence of glucose, explain/describe what would happen, for each mutant, to the expression of the lac operon when you add lactose AND what would happen when the bacteria has used up all of the lactose (if the mutant is able to use lactose).5. Mutations in the lac operator that strengthen the binding of the lac repressor 200 fold 6. Mutations in the promoter that prevent binding of RNA polymerase 7. Mutations in CRP/CAP protein that prevent binding of cAMP8. Mutations in sigma factor that prevent binding of sigma to core RNA polymerasearrow_forwardMolecular Biology Please help and there is an attached image. Thank you. A bacteria has a gene whose protein/enzyme product is involved with the synthesis of a lipid necessary for the synthesis of the cell membrane. Expression of this gene requires the binding of a protein (called ACT) to a control sequence (called INC) next to the promoter. A. Is the expression/regulation of this gene an example of induction or repression?Please explain:B. Is this expression/regulation an example of positive or negative control?C. When the lipid is supplied in the media, the expression of the enzyme is turned off.Describe one likely mechanism for how this “turn off” is accomplished.arrow_forward
arrow_back_ios
SEE MORE QUESTIONS
arrow_forward_ios
Recommended textbooks for you
- Surgical Tech For Surgical Tech Pos CareHealth & NutritionISBN:9781337648868Author:AssociationPublisher:CengageCase Studies In Health Information ManagementBiologyISBN:9781337676908Author:SCHNERINGPublisher:Cengage
Surgical Tech For Surgical Tech Pos Care
Health & Nutrition
ISBN:9781337648868
Author:Association
Publisher:Cengage
Case Studies In Health Information Management
Biology
ISBN:9781337676908
Author:SCHNERING
Publisher:Cengage