The process involved in evolutionary modification of ectodermal appendages into the sweat glands. Also explain the evidence which indicates that sweat gland is a modification of hair, which develops at the expense of hair follicle. Introduction: The analysis of ectodermal appendages shows that humans are indistinguishable in terms of hair follicle density when compared to other primate species like chimpanzees. The shedding of hairy coats by the early humans related to their closest evolutionary ancestors has been evaluated with the emergence of sweat glands, the tiny glands which allow the body to cool down rapidly. The presence of sweat gland makes human fit to survive in a wide variety of climates. The studies based on the correlation of definitive hair and eccrine gland density traits of humans and our closely related primate species help in the elucidation of genetic changes that leads to this emergence and drove the adaptive changes.

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Chapter 16, Problem 3DQ

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The process involved in evolutionary modification of ectodermal appendages into the sweat glands. Also explain the evidence which indicates that sweat gland is a modification of hair, which develops at the expense of hair follicle.

Introduction:

The analysis of ectodermal appendages shows that humans are indistinguishable in terms of hair follicle density when compared to other primate species like chimpanzees. The shedding of hairy coats by the early humans related to their closest evolutionary ancestors has been evaluated with the emergence of sweat glands, the tiny glands which allow the body to cool down rapidly. The presence of sweat gland makes human fit to survive in a wide variety of climates. The studies based on the correlation of definitive hair and eccrine gland density traits of humans and our closely related primate species help in the elucidation of genetic changes that leads to this emergence and drove the adaptive changes.

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*Example 2: Tracing the path of an autosomal dominant trait Trait: Neurofibromatosis Forms of the trait: The dominant form is neurofibromatosis, caused by the production of an abnormal form of the protein neurofibromin. Affected individuals show spots of abnormal skin pigmentation and non-cancerous tumors that can interfere with the nervous system and cause blindness. Some tumors can convert to a cancerous form. i The recessive form is a normal protein - in other words, no neurofibromatosis.moovi A typical pedigree for a family that carries neurofibromatosis is shown below. Note that carriers are not indicated with half-colored shapes in this chart. Use the letter "N" to indicate the dominant neurofibromatosis allele, and the letter "n" for the normal allele. Nn nn nn 2 nn Nn A 3 N-