Bundle: Human Biology, Loose-leaf Version, 11th + MindTap Biology, 1 term (6 months) Printed Access Card
Bundle: Human Biology, Loose-leaf Version, 11th + MindTap Biology, 1 term (6 months) Printed Access Card
11th Edition
ISBN: 9781305616660
Author: Cecie Starr, Beverly McMillan
Publisher: Cengage Learning
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Chapter 15, Problem 4CT

Secretion of the hormone ADH may decrease or stop if the pituitary’s posterior lobe is damaged, as by a blow to the head. This is one cause of diabetes insipidus. People with this form of diabetes excrete so much dilute urine that they may become seriously dehydrated. Where are the target cells of ADH?

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Secretion of the hormone ADH may decrease or stop if the pituitary’s posterior lobe is damaged, as by a blow to the head. This is one cause of diabetes insipidus. People with this form of diabetes excrete so much dilute urine that they may become seriously dehydrated. Where are the target cells of ADH?
The kidneys are important regulators of blood volume (and thus MAP) and the ionic composition and pH of plasma . The amount of water reabsorbed or lost is hormonally regulated. Both the amount of blood that is filtered (glomerular filtration rate) and the amount of water that is reabsorbed is determined by hormones binding with receptors. In order to understand why the level of any given hormone is elevated or low in the blood, it is necessary to think in terms of homeostasis loops for each hormone, and what the initial disturbance to homeostasis was. In other words “who started it”! What STEROID hormone is elevated by increased activation of the RAS pathway?
The kidneys are important regulators of blood volume (and thus MAP) and the ionic composition and pH of plasma . The amount of water reabsorbed or lost is hormonally regulated. Both the amount of blood that is filtered (glomerular filtration rate) and the amount of water that is reabsorbed is determined by hormones binding with receptors. In order to understand why the level of any given hormone is elevated or low in the blood, it is necessary to think in terms of homeostasis loops for each hormone, and what the initial disturbance to homeostasis was. In other words “who started it”! What NON STEROID hormone is produced at the end (but as part of) of the RAS pathway?
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