It would inhibit muscarinic receptorsO It would cause excess sweatingO It would constrict the airwaysIt would raise blood pressureO It would laste yucky Why should someone with an obstructive lung disease not be prescribed beta blockers?It would inhibit muscarinic receptorsIt would cause excess sweatingIt would constrict the airways

A chronic inflammatory lung disease that causes hindered airflow from the lungs is called as Chronic…

Answered: Why should someone with an obstructive…

Human Anatomy & Physiology (11th Edition)

11th Edition

ISBN:9780134580999

Author:Elaine N. Marieb, Katja N. Hoehn

Publisher:Elaine N. Marieb, Katja N. Hoehn

Chapter1: The Human Body: An Orientation

Section: Chapter Questions

Problem 1RQ: The correct sequence of levels forming the structural hierarchy is A. (a) organ, organ system,...

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Similar questions

Why muscarinic receptor antagonists are different from adrenergic beta-2-receptor agonists. What is the major benefit of using adrenergic beta-2-receptor agonists?
Select all the second messengers
Patients with a history of heart failure often get this history from chronic hypertension. Chronic hypertension leads to a condition called hypertrophic heart disease, or hypertrophic cardiomyopathy. This means that the heart muscle gets bigger, allowing less room for it to fill with blood. If these patients take beta blockers, such as metoprolol, it will benefit them because the heart will not overwork and grow in size. However, if a patient has pulmonary edema, how could the beta blocker make this patient worse?
What are the superiorities of PPI over H2 blocker? How Omeprazole acts? Please briefly explain at your own words.
In which of the following situations is calcium moving through a channel in the pacemaker membrane driven by concentration but against charge? O during the repolarization phase of the action potential, just before the channel closes O during the depolarization phase of the action potential, just after the channel opens O never O at all times that sodium is moving through the membrane O during the repolarization phase of the action potential, just after the channel opens O during the depolarization phase of the action potential, just before the channel closes O during the pacemaker potential, just before the funny channel closes
1. List the specific steps of beta-adrenergic receptor signaling through PKA activation. 2. What is the chromophore in rhodopsin? How does light absorption activate rhodopsin signaling? 3. How does the rhodopsin signaling pathway effector enzyme PDE affect levels of cGMP? At the molecular level, what is the result of the changed levels of cGMP and what response does this trigger? 4. What class of receptor is the insulin receptor (INSR)? When IRS1 is phosphorylated by the insulin receptor, what are the two signaling pathway branches that are activated? What physiological responses do these two branches regulate? 5. Both insulin and epinephrine regulate levels of D-glucose in the blood. How?
When a patient has a severe allergic reaction, a common prescribed drug is epinephrine. Which of the follow best explains the effects of epinephrine on a patient experiencing a severe allergic reaction? A epinephrine binds to the beta-1 receptor. Activating the beta-1 receptor causes vasodilation and bronchoconstriction. Bronchoconstriction allows the patient to breath by relaxing the smooth muscle that is constricting the airway. B Epinephrine binds to the beta-2 receptor. Activating the beta-2 receptor causes vasodilation and bronchodilation. Bronchodilation allows the patient to breath easier by relaxing the smooth muscle that is constricting the airway. C Epinephrine binds to the alpha-1 receptor. Activating the alpha-1 receptor causes vasodilation and bronchodilation. Bronchodilation allows the patient to breath by relaxing the smooth muscle that is constricting the airway. D Epinephrine binds to the beta-1 receptor. Activating the beta-1 receptor causes vasodilation and…
Medical practice is paying more attention to theautonomic system in considering disease states. Whywould autonomic tone be important in consideringcardiovascular disease?
There is an animal that uses almost 100 percent of the oxygen in its blood. Analyzing the Hb of this animal, it’s found that 2,3-BPG does not bind to Hb, but bicarbonate ions do. Knowing that bicarbonate ions are strong negative allosteric effectors, what do you make of these findings? Why is the animal’s Hb not responsive to 2,3 BGP, but responsive to bicarbonate ions?

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Why should someone with an obstructive lung disease not be prescribed beta blockers? O It would inhibit muscarinic receptors O It would cause excess sweating OIt would constrict the airways