Which of the following is NOT an example of non-specific (innate) resistance mechanisms interacting directly with specific acquired immunity mechanisms? what option below is the answer? Antibodies binding to bacterial surface antigens, coating (opsonizing) the bacteria and enhancing their phagocytosis by leukocytes Macrophages and dendritic cells presenting antigens to lymphocytes None of the other four answers (All are examples of interaction between innate and acquired resistance) Antibody-dependent cell-mediated cytotoxicity Complement being activated by antigen-antibody binding
Which of the following is NOT an example of non-specific (innate) resistance mechanisms interacting directly with specific acquired immunity mechanisms? what option below is the answer? Antibodies binding to bacterial surface antigens, coating (opsonizing) the bacteria and enhancing their phagocytosis by leukocytes Macrophages and dendritic cells presenting antigens to lymphocytes None of the other four answers (All are examples of interaction between innate and acquired resistance) Antibody-dependent cell-mediated cytotoxicity Complement being activated by antigen-antibody binding
Human Anatomy & Physiology (11th Edition)
11th Edition
ISBN:9780134580999
Author:Elaine N. Marieb, Katja N. Hoehn
Publisher:Elaine N. Marieb, Katja N. Hoehn
Chapter1: The Human Body: An Orientation
Section: Chapter Questions
Problem 1RQ: The correct sequence of levels forming the structural hierarchy is A. (a) organ, organ system,...
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Which of the following is NOT an example of non-specific (innate) resistance mechanisms interacting directly with specific acquired immunity mechanisms?
what option below is the answer?
Antibodies binding to bacterial surface antigens, coating (opsonizing) the bacteria and enhancing their phagocytosis by leukocytes
Macrophages and dendritic cells presenting antigens to lymphocytes
None of the other four answers (All are examples of interaction between innate and acquired resistance)
Antibody-dependent cell-mediated cytotoxicity
Complement being activated by antigen-antibody binding
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