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Which of the following is a mechanism through which pain signals are modulated? O the pain and temperature afferents are all unmyelinated O pain fibers only have a rapid response to pain that dulls quickly O acetylcholine release from the dorsal raphe reduces the response to pain receptors for touch provide inhibitory inputs to pain fibers in the dorsal horn

Which of the following is a mechanism through which pain signals are modulated? O the pain and temperature afferents are all unmyelinated O pain fibers only have a rapid response to pain that dulls quickly O acetylcholine release from the dorsal raphe reduces the response to pain receptors for touch provide inhibitory inputs to pain fibers in the dorsal horn

Human Anatomy & Physiology (11th Edition)
Human Anatomy & Physiology (11th Edition)
11th Edition
ISBN:9780134580999
Author:Elaine N. Marieb, Katja N. Hoehn
Publisher:Elaine N. Marieb, Katja N. Hoehn
Chapter1: The Human Body: An Orientation
Section: Chapter Questions
Problem 1RQ: The correct sequence of levels forming the structural hierarchy is A. (a) organ, organ system,...
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**Mechanisms of Pain Signal Modulation** A comprehensive understanding of how pain signals are modulated can help us in managing and treating pain more efficiently. Below are some mechanisms through which pain signals are modulated: 1. **The Pain and Temperature Afferents are all Unmyelinated** - Pain and temperature signals are often carried by unmyelinated fibers, which conduct signals more slowly. This slower conduction is thought to play a role in the modulation of pain signal transmission. 2. **Pain Fibers Only Have a Rapid Response to Pain That Dulls Quickly** - Certain pain fibers, known as A-delta fibers, are responsible for the rapid transmission of sharp, acute pain which typically dulls quickly after the initial injury. This quick dulling of pain is a form of modulation. 3. **Acetylcholine Release from the Dorsal Raphe Reduces the Response to Pain** - The release of neurotransmitters such as acetylcholine from the dorsal raphe nucleus can interact with systems in the spinal cord to reduce pain signals, providing an inhibitory effect on pain transmission. 4. **Receptors for Touch Provide Inhibitory Inputs to Pain Fibers in the Dorsal Horn** - The dorsal horn of the spinal cord contains receptors for touch that can inhibit pain fibers. This is why rubbing a painful area can sometimes alleviate pain; the touch receptors interfere with pain signal transmission. Understanding these mechanisms is crucial for developing new pain management therapies and improving existing ones.
Transcribed Image Text:**Mechanisms of Pain Signal Modulation** A comprehensive understanding of how pain signals are modulated can help us in managing and treating pain more efficiently. Below are some mechanisms through which pain signals are modulated: 1. **The Pain and Temperature Afferents are all Unmyelinated** - Pain and temperature signals are often carried by unmyelinated fibers, which conduct signals more slowly. This slower conduction is thought to play a role in the modulation of pain signal transmission. 2. **Pain Fibers Only Have a Rapid Response to Pain That Dulls Quickly** - Certain pain fibers, known as A-delta fibers, are responsible for the rapid transmission of sharp, acute pain which typically dulls quickly after the initial injury. This quick dulling of pain is a form of modulation. 3. **Acetylcholine Release from the Dorsal Raphe Reduces the Response to Pain** - The release of neurotransmitters such as acetylcholine from the dorsal raphe nucleus can interact with systems in the spinal cord to reduce pain signals, providing an inhibitory effect on pain transmission. 4. **Receptors for Touch Provide Inhibitory Inputs to Pain Fibers in the Dorsal Horn** - The dorsal horn of the spinal cord contains receptors for touch that can inhibit pain fibers. This is why rubbing a painful area can sometimes alleviate pain; the touch receptors interfere with pain signal transmission. Understanding these mechanisms is crucial for developing new pain management therapies and improving existing ones.
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