What are the three reasons given why aging can’t be modulated?

Alzheimer's disease is a progressive neurological degenerative disorder that affects almost 50 million people worldwide. The cause of Alzheimer's disease is still not well understood. It is thought to be a combination of genetics, lifestyle, and environmental causes. It is also difficult to diagnose Alzheimer's disease during the initial stages of the disease as symptoms can be mistaken for normal aging. Currently, there is no cure for the disease. Medication can help manage the symptoms but degeneration of brain tissue cannot be stopped or reversed. One medication used to manage the symptoms of Alzheimer's disease is a cholinesterase inhibitor. The medication helps to improve brain functioning and memory.   What is the function of cholinesterase in the synapse? How does a cholinesterase inhibitor work in a synapse?  Explain the function of cholinesterase inhibitor in increasing brain functioning and memory

Long-term stress and sleep deprivation is associated with memory loss and negative thoughts.Subjects in a research study were shown images after they received either a full night’s rest or limited rest. The subjects were shown negative, positive, and neutral images. The subjects with limited rest remembered most of the negative images but only half of the positive and neutral images.The structures of the brain affected by long-term stress and sleep deprivation are, respectively: Select one: a. Long-Term Stress Sleep Deprivation Temporal lobe Frontal lobe b. Long-Term Stress Sleep Deprivation Temporal lobe Parietal lobe c. Long-Term Stress Sleep Deprivation Frontal lobe Occipital lobe d. Long-Term Stress Sleep Deprivation Frontal lobe Parietal lobe

Can you still have repeats of CAG without having Huntington’s Disease? AND Can you think of any other disease that have quite a few of the same symptoms as Huntington’s Disease?

Alzheimer's Disease  Alzheimer's disease is a progressive neurodegenerative disorder that affects almost 50 million people worldwide. The cause of Alzheimer's disease is still not well understood. It is thought to be a combination of genetics, lifestyle, and environmental causes. It is also difficult to diagnose Alzheimer's disease during the initial stages of the disease as symptoms can be mistaken for normal ageing. Currently, there is no cure for the disease. Medication can help manage the symptoms but degeneration of brain tissue cannot be stopped or reversed.  Currently, the medication that manages the symptoms of Alzheimer's disease is a type of cholinesterase inhibitor. In other words, the medication inhibits the function of cholinesterase and helps in brain functioning and memory.  What is the function of cholinesterase in the synapse?  How does a cholinesterase inhibitor work in a synapse?  Explain how cholinesterase inhibitors are able to increase brain functioning and…

Which phase of the general adaptation syndrome occurs over a longer period of time and how?

What is the most environmental cue for entrainment of circadian rhythms?

The development of Alzheimer's disease is not a normal part of aging, although a person’s risk of developing the condition increases after age 65. Approximately half of people over age 85 have Alzheimer's disease. In cases of rare genetic changes that lead to Alzheimer's disease, symptoms present themselves in the 40s or 50s. For this essay considering the importance of education and the prevention of Alzheimer's Disease Question With reasons, pointing at the target audience with their age range, sex, ethnicity, describe what information and the method of delivery you would use basing on their importance and why?

In Huntington’s disease........... cells of the.................. and.......... pathways degenerate resulting in  movements

Electroconvulsive therapy (ECT) is a reasonable option for intractable depression or schizophrenia, but a mechanism of action has not been established. One credible hypothesis is related to neural plasticity. Below is the neuronal gene expression assay for four genes after ECT, normalized with the Hamilton depression rating scale of 21 items version (HAM-D-21). Which of the following conclusion you may NOT conclude from the data? ECT may convert adult neurons into IPSCs which may divide and differentiate into healthy adult neurons Neural plasticity steadily increases with the number of ECT administration ECT seems to increase the expression of Yamanaka’s four transcription factors ECT may increase neural plasticity