Two men are diagnosed with diabetes insipidus. One didn’t have the disorder until he suffered a stroke. The other had a withstood the condition all his life, and it had never responded to exogenous ADH despite the presence of normal ADH receptors. What might be the cause of the diabetes insipidus in the two men?
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1. Two men are diagnosed with diabetes insipidus. One didn’t have the disorder until he suffered a stroke. The other had a withstood the condition all his life, and it had never responded to exogenous ADH despite the presence of normal ADH receptors. What might be the cause of the diabetes insipidus in the two men?
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- Lifestyle modification could be very useful for this patient. What would these modificationsinclude and would they help resolve the type 1, type 2, or both types of diabetes mellitus that this patient is experiencing? Explain your answer.Endocrine Can I get the answer? I do not need explanation. 1. Coronary artery disease is the leading cause of death associated with diabetes mellitus. True False 2. Insulin Glargine is a long-acting form of insulin that is synthesized with several D-amino acids that slow its proteolytic degradation and extend the half-life of the insulin Glargine molecule. True False 3. Metformin is a drug that lowers blood glucose levels by increasing glucose uptake in skeletal muscle, blocking the hepatic production of glucose, and decreasing the intestinal absorption of glucose. True FalseHi, can you help me with the following multiple choice questions: Thanks!! Z. is a 27-year-old man with readily apparent thyroid goiter. He comments that he gained 5 pounds in the last year, and you notice that his weight is approximately 20 pounds greater than normal. What can you conclude about this patient’s thyroid function? the goiter indicates that he is hyperthyroid the combination of excessive weight and goiter indicates that he is hypothyroid the combination of excessive weight and goiter indicates that he is hyperthyroid he is probably euthyroid, because the weight gain eliminates the possibility of hyperthyroidism it is not possible to make a conclusion from this information provided. For an answer to be correct, both the symptom and the explanation must be correct. Which answer is incorrect? the resting heart rate increases because circulating levels of catecholamines increase. Excess thyroid hormones stimulate the release of more adrenal catecholamines. myocardial…
- Match the following. Place letter answer in space provided at left. Answers may be used more than once or not at all 1. A second messenger A. ADH 2. chemical needed to convert glucose to ATP B. Antagonistic 3. chemicals that exert effects on the same cells that secrete them C. autocrines 4. Effect of an increase in one hormone causing a decrease in another D. Calcitonin 5. Effect of an increase in one hormone causing an increase in another E. Corpus luteum 6. Effect where hormones are released via nerve fibers F. DAG 7. Hormone responsible for the deposition of calcium G. FSH 8. hormone types that has direct gene activation H. Glycogen 9. Hypophyseal hormone released in response to low sugar levels I. HGH 10. Identify generically the first messenger J. Hormone 11. Identify the enzyme involved in PIP calcium signaling K. Liver 12. identify the hypothalamic hormone that regulates serum water levels L. Neuronal 13. identify the hypothalamic hormone that aids in milk ejection M. None of…I am struggling with deciding what adrenal eitologies line up best with these case studies. A 22 year-old woman (no family history available, not currently taking medication, negative medicalhistory) presents with.... C. mild hypotension and hyperkalemia. Blood pH is 7.38 and BUN is 34mg/dL and GFR is 82ml/min. D. hypotension with muscle weakness, fatigue, weight loss, heart palpitation, weight loss,and secondary amenorrhea. Her laboratory results reveal hyperkalemia. Fasting bloodglucose is 55mg/dL, serum albumin is 8.0 mg/dL and blood pH is 7.35. E. hypertension with headache, diarrhea and abdominal pain. The patient also suffersfrom panic attacks and hot flashes. Fasting plasma glucose is 285 mg/dL and TSH is 6.5μU/mL.Type- 2 diabetes mellitus is: 1.both a secondary pathology and usually the result of inappropriate hormone secretion 2.both a secondary pathology and usually the result of abnormal target cell responsiveness 3.usually the result of inappropriate hormone secretion 4.a secondary pathology 5.usually the result of abnormal target cell responsiveness
- Question is below:- A patient has a brain tumor that necessitates removal of his pituitary gland. Will its removal affect production of ADH and oxytocin? PleaseExplain..?Escalation of care is actioned/intervened for dehydrated patients with tye 1 diabetes. Why?Answer the following: 1.) an indirect agonist that mimics the action of epinephrine and norepinephrine and either stimulate release or block the reuptake of the naturally occuring sympathomimetics 2.) Toxic concentration of alprazolam 3. The only approved indications for use of this drug is for the treatment of attention-deficit hyperactivity disorder (ADHD) 4. An elevated acetylsalicylic acid serum concentration will decrease serum pH and contributes to 5. the metabolism of acute salicylate poisoning follows what order of metabolism kinetic? 6. In TCA poisoning, why is hemodialysis not recommended as a way of treating patient? 7. after an IV administration of this drug an individual feels a pleasurable experience 8, First generation of the tricyclic antidepressant agents 9. An antidote of choice for iron overdose/poisoning 10. Pediatric toxic dose of salicylates 11. Lethal dose of Amphetamine for children
- Answer the following in 2-3 sentences. 1. What effect does insulin have on glycogen metabolism? 2. What effect does glucagon have on blood glucose level? 3. What organ is the source of insulin and glucagon? 4. The hormone epinephrine generates a second messenger. Explain 5. Compare the target tissues for glucagon and epinephrine.Diabetes insipidus has been identified in two males. One person did not have the condition until he had a stroke. The other had lived with the condition his whole life, and despite the existence of normal ADH receptors, it had never reacted to exogenous ADH. What might be the cause of the two men's diabetes insipidus? Provide your references.Kindly answer all the questions below. These are all the questions left.8. What is the function of aldosterone?a. Regulate the concentration of sodium ions in the urine. b. Affect sexual characteristics.c. Regulate the release of glucose from the liver.d. Regulate fatty acid release from the adipose tissue.9. How do peptide hormones affect a target cell’s transcription?a. They bind to the receptor on plasma membrane and initiate a signaling pathway that regulates transcriptions. b. They bind to the plasma membrane get taken inside and bind to the DNA.c. They enter the cell and bind with a receptor in the cytoplasm then bind to the DNA. d. They enter the cell and bind with a receptor in the nucleus on the DNA.10. What sequence describes how a steroid hormone impacts gene expression?a. Steroid hormone crosses the plasma membrane, binds to an intracellular hormone receptor in the nucleus, then regulates transcription. b. Steroid hormone binds with a plasma membrane receptor, then…