Answered: The protein on the right called…

The protein on the right called desmolaris, also called Draculin, found in bats. It is a non competitive inhibitor of Factor Xa. So what is this protein for? Why inhibit Factor Xa?

Biochemistry

9th Edition

ISBN:9781319114671

Author:Lubert Stryer, Jeremy M. Berg, John L. Tymoczko, Gregory J. Gatto Jr.

Publisher:Lubert Stryer, Jeremy M. Berg, John L. Tymoczko, Gregory J. Gatto Jr.

Chapter1: Biochemistry: An Evolving Science

Section: Chapter Questions

Problem 1P

See similar textbooks

Similar questions

In the normal cycle of Ras activity, when Ras is bound to y activity that removes a phosphate to convert GTP back to GDP and this it is inactive. Activation of Ras involves exchanging GDP for The Ras protein has intrinsic Ras. inactivates activates ATP GTP «< Questic A Moving to another question will save this response. ADP GDP GTPase Clo nuclease
For the protein Calmodulin-1: 1) this protein have any secondary, tertiary and/or quaternary structures present? 2) Are there any molecular additions to this protein that are not amino acids?For both questions, can you explain if so, where specifically? The protein Calmodulin-1 is stated to have the molecular function of signaling receptor binding which is interrelated with the biological function of calcium-mediated signaling. Can you explain this relationship?
Many patients become resistant to HIV protease inhibitors with the passage of time, owing to mutations in the HIV gene that encodes the protease. Mutations are not found in the aspartate residue that interacts with the drugs. Why not?
please identify amino acids in the N terminus in the picture below.
The seed of the castor bean (Ricinus communis) contains large amounts of ricin, a deadly poison for animals, including humans. One of the two subunits of this toxin is a lectin that binds terminal N-acetylgalactosamine residues on glycoproteins on the surface of eukaryotic cells, allowing the other subunit to enter the cell and kill it by preventing proteins from being made. Select the possible antidotes that could prevent or reverse ricin-mediated entry of the toxin. a high concentration of N-acetylgalactosamine an oligosaccharide with the same structure as that recognized by ricin a compound that prevents synthesis of extracellular glycoproteins a compound that activates enzymes that break down glycogen into glucose an enzyme that degrades lipopolysaccharides
Interleukin-6 is an important pro-inflammatory cytokine released in response to a variety of infections. Tocilizumab is a monoclonal antibody that blocks the action of IL-6. This drug cannot be given orally. Provide a biochemical explanation for this.
The action of this enzyme found in Beano alpha-galactosidase can also according to many sources be used to remove ____________ from the red blood cells of the B blood group and convert them to the O blood group. Since the _________ molecules (composed of many monomers) are connnected to protein on the membrane and also the cell membrane of the red blood cell itself; therefore these molecules help create cell identity and recognition, and when the are connected to proteins they are known as _________ , but if they are connected to the cell membrane itself they are known as ____________. This is a good question for reviewing the structures on the cell membrane as well. The only difference between the B-blood type and the O-blood type is ________. The following is a diagram of the differences of these molecules that determine blood type. List of answers: disaccharid, proteins ,dehydration synthesis ,nucleotide ,amino acids ,nucleic acids ,lactose ,fructose ,sucrose ,hydrolysis…
why does the activation of M-cdk also result in the activation of Cdc25? What is the mechanism behind it?
In 1994, the U.S. Public Health Service identified “10 Essential Public Health Services.” For each of the ten, please briefly describe a specific activity that has been used in the response to COVID-19.
Could you please help answer this question?
What is the mechanism of the Contactin-BIG-1
. Studies on CAMP actions in cultured cells usually involve adding to the cell culture not CAMP, but dibutyryl CAMP (see structure). Why is this structural modification necessary? How could you test the premise that di-Bu-CAMP has the same biochemical effects as CAMP? N. N= Nat Dibutyryl CAMP O