Incorrect amino acid changes: Justin (CGC--> AGC) Patient A (GGA-->GAA) Patient B (CGG-->CCG) Patient C (CCT-->TCT) What amino acid changes are associated with the enzymes from the other three patients and how would changes lead to non-functional enzymes?
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Incorrect amino acid changes:
Justin (CGC--> AGC)
Patient A (GGA-->GAA)
Patient B (CGG-->CCG)
Patient C (CCT-->TCT)
What amino acid changes are associated with the enzymes from the other three patients and how would changes lead to non-functional enzymes?
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- A 9 year old mentally retarded girl with a protuberant abdomen, short stature, coarse facial features and cloudy corneas. skeletal malformations include dysostosis Multiplex and Bullet shaped middle phalanx .what is the enzyme deficient in this patient? A)Iduronate sulfatase B) beta - Galactosidase C)alpha - L- Iduronidase D) beta - GlucuronidaseSerum blood of a patient with dislipoproteinemia type 1 has milky appearance even in fasting. If serum stays at low temperature (40) for several hours fatty layer appears on its surface. What are the possible causes of these symptoms? To explain this, answer the questions and do the following tasks: a) what compounds of serum must be tested for that patient in biochemical lab? b) write the reaction which does not occur properly in patient’s blood; c) write down the schemes, explaining how the products of the previous reaction are used in adipose tissue and heart in healthy person 2 hours after a meal.explain the phenylketonuria disease mechanism
- What step ends the first-half of the chymotrypsin mechanism? Attack of water on the acyl-enzyme intermediate. Selected Collapse of the first tetrahedral intermediate, releasing of the C-terminal peptide fragment and formation of the acyl-enzyme intermediate. Deprotonation of serine by the histidine. Collapse of the second tetrahedral intermediate leading to the final products. REVIEW QUESTION 1/1 CONTINUE MacBook Pro https://virginiacommonwealth.instructure.com/courses/42082/assignments/271397 esc OTWhat is the mechanism of the reaction of the Bradford reagent with proteins? (explain in not less than 5 sentences).discuss the metabolic defects that causes phenylketonuria and the strategies used to diagnose and treat this condition.
- Now answer the following questions: 1. Is this patient experiencing a disorder affecting anaerobic or aerobic metabolism? a) disorder affecting anaerobic metabolism. b) disorder affecting aerobic metabolism. c) None of them d) Both 2. You decide to perform assays to check the activity of one or more metabolic enzymes in the red blood cells. Which enzyme(s) would you check? a) Amylase b) Enzymes of pentose phosphate pathways c) Transaminase enzymes d) hexokinase, phosphofructokinase, and pyruvate kinaseOn the right is a diagram of plasma concentration of three metabolites: glucose, B- hydroxybutyrate, and free fatty acid of an experimental animal over weeks of starvation. 4. (a) Label the lines with the names of the three metabolites. Yellow: Red: 10 20 30 40 Blue: Days of starvation (b) What factor in the plasma binds to and transports free fatty acid (c) Where was B-hydroxybutyrate produced? Why it is produced? What are the purposes of producing it (d) How B-hydroxybutyrate is used as fuel in extrahepatic cells? Write down the reactions, enzymes, and cofactors (No need to elaborate on the TCA cycle). (e) How many ATP molecules or equivalent are generated after complete oxidation of one molecule of B-hydroxybutyrate? Show your calculations. Hint: You need to consider ATP consumption for bypassed steps that may normally yield or consume AT Plasma concentration (mM)What is the medical diagnostic value associated with the presence of the following enzyme in the bloodstream? ALT
- Describe type I, II, and III L-asparaginases. Mention characteristics and differences between them with respect to enzyme kinetics (rate, km, etc).A 38-year-old woman went to a hospital and complained of a non-productive cough and dyspnea which has progressed over two weeks. Based on physical examination, she was pale, diaphoretic, and in acute respiratory distress. According to her, she had sexual contact with his partner for the past weeks. The physician ordered laboratory tests and results revealed that the CD4 count was significantly low. A. What enzymes might elevate and decrease in concentration based on the diagnosis of the patient and why?B. What other laboratory tests in the clinical chemistry section will correspond to the patient’s diagnosis?Why were the outcomes so different when reduced ribonuclease was reoxidized in the presence and absence of urea?