diagram shows an internal Teedback hism that helps the body maintain non stasis. Homeostasis in the Body Resea hers teste sugar levels of 100 volunteers exactly two hours after eating a specific meal. Based on their ages, the volunteers were expected to have a healthy blood sugar level of less than 140 mg/dL. Approximately 10% of the volunteers had blood sugar levels higher than 140 mg/dL. Using the diagram, infer one possible cause for these high blood sugar levels. Raises blood sugar High blood sugar Glycogen Liver Stimulates glycogen breakdown Glucose Glucagon Lowers blood sugar Stimulates glycogen formation Insulin 0000 Tissue cells D Stimulates glucose uptake from blood Promotes insulin release Pancreas Promotes glucagon release Low blood sugar O The liver was not responsive to the glucagon that was produced. O The body did not produce enough insulin to have the expected effect. O The pancreas stopped glucagon production. The pancreas produced too much insulin.

Human Anatomy & Physiology (11th Edition)
11th Edition
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Chapter1: The Human Body: An Orientation
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The diagram illustrates an internal feedback mechanism crucial for maintaining homeostasis in the body, specifically focusing on blood sugar regulation. Researchers tested the blood sugar levels of 100 volunteers precisely two hours post-meal. Based on age groups, a healthy blood sugar level was expected to be under 140 mg/dL. However, approximately 10% of the volunteers exhibited levels exceeding 140 mg/dL.

### Diagram Explanation

#### Components:

1. **Liver**:
   - Stores Glycogen.
   - Releases Glucose.

2. **Pancreas**:
   - Produces Insulin and Glucagon.
   - Promotes Insulin release to lower blood sugar.
   - Promotes Glucagon release to raise blood sugar.

3. **Tissue Cells**:
   - Stimulates glucose uptake from blood (aided by Insulin).

#### Processes:

- **High Blood Sugar**:
  - Prompts the pancreas to release Insulin.
  - Insulin facilitates glucose uptake by tissue cells.
  - Stimulates glycogen formation in the liver.
  
- **Low Blood Sugar**:
  - Stimulates the pancreas to release Glucagon.
  - Glucagon triggers glycogen breakdown in the liver, increasing blood sugar levels.

### Inference Options:

1. **The liver was not responsive to the glucagon that was produced.**
2. **The body did not produce enough insulin to have the expected effect.**
3. **The pancreas stopped glucagon production.**
4. **The pancreas produced too much insulin.**

These options suggest potential causes for high blood sugar levels observed in some volunteers. Understanding these mechanisms is key to diagnosing and managing blood sugar-related issues, such as diabetes.
Transcribed Image Text:The diagram illustrates an internal feedback mechanism crucial for maintaining homeostasis in the body, specifically focusing on blood sugar regulation. Researchers tested the blood sugar levels of 100 volunteers precisely two hours post-meal. Based on age groups, a healthy blood sugar level was expected to be under 140 mg/dL. However, approximately 10% of the volunteers exhibited levels exceeding 140 mg/dL. ### Diagram Explanation #### Components: 1. **Liver**: - Stores Glycogen. - Releases Glucose. 2. **Pancreas**: - Produces Insulin and Glucagon. - Promotes Insulin release to lower blood sugar. - Promotes Glucagon release to raise blood sugar. 3. **Tissue Cells**: - Stimulates glucose uptake from blood (aided by Insulin). #### Processes: - **High Blood Sugar**: - Prompts the pancreas to release Insulin. - Insulin facilitates glucose uptake by tissue cells. - Stimulates glycogen formation in the liver. - **Low Blood Sugar**: - Stimulates the pancreas to release Glucagon. - Glucagon triggers glycogen breakdown in the liver, increasing blood sugar levels. ### Inference Options: 1. **The liver was not responsive to the glucagon that was produced.** 2. **The body did not produce enough insulin to have the expected effect.** 3. **The pancreas stopped glucagon production.** 4. **The pancreas produced too much insulin.** These options suggest potential causes for high blood sugar levels observed in some volunteers. Understanding these mechanisms is key to diagnosing and managing blood sugar-related issues, such as diabetes.
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