R1 and Su B mutations are intragenic suppressors or intergenic suppressors. Introduction: Person B and Person C conducted nonsense mutation on E. coli strain. During mutation, the structural gene encodes an enzyme called L-ribulokinase that allows the bacteria to metabolize arabinose sugar. The revertants of mutant strain showed two different suppressor mutation R1 and Su B . R1 is the original mutation in L-ribulokinase and not able to metabolize arabinose sugar as the enzyme encoded by the wild type allele. The second suppressor mutation Su B is not linked to the original mutation. However, the second suppressor able to produce L-ribulokinase in conjunction with R1 mutation

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Answer 1

Question

Chapter 18.1, Problem 45CQ

a.

Summary Introduction

To determine:

R1 and Su^B mutations are intragenic suppressors or intergenic suppressors.

Introduction:

Person B and Person C conducted nonsense mutation on E. coli strain. During mutation, the structural gene encodes an enzyme called L-ribulokinase that allows the bacteria to metabolize arabinose sugar. The revertants of mutant strain showed two different suppressor mutation R1 and Su^B. R1is the original mutation in L-ribulokinase and not able to metabolize arabinose sugar as the enzyme encoded by the wild type allele. The second suppressor mutation Su^B is not linked to the original mutation. However, the second suppressor able to produce L-ribulokinase in conjunction with R1 mutation

b.

Summary Introduction

To explain:

The way in which R1 and Su^B restore the ability of araB14 to metabolize arabinose and why Su^B is able to more fully restore this ability.

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a. What percentage of a drug is eliminated after 4 half-lives? Please round to the nearest percent. b. What will happen to elimination of the drug in the previous question if the system is saturated? explain and show any math involved

If you wanted to reduce the difference between peak and trough levels that occur with repeated administration of a drug, how would you adjust the dose and dose interval without changing the plateau concentration (plateau is the average of peak and trough levels)? Select your answers for both dose and interval. Hint: It may be helpful to think about this problem using an example such as food. How would you eat if you wanted to maintain very steady hunger/satiety levels without changing your total caloric intake? Options: A. Dose; Increase dose B. Dose; Decrease dose C. Dose; Do not change dose D. Interval; Increase the interval between doses (give the drug less frequently) E. Interval; Decrease the interval between doses (give the drug more frequently) F. Interval; Do not change the interval

What percentage of a drug is eliminated after 4 half-lives? Please round to the nearest percent. Show the math

Answer 2

Question

Chapter 18.1, Problem 45CQ

a.

Summary Introduction

To determine:

R1 and Su^B mutations are intragenic suppressors or intergenic suppressors.

Introduction:

Person B and Person C conducted nonsense mutation on E. coli strain. During mutation, the structural gene encodes an enzyme called L-ribulokinase that allows the bacteria to metabolize arabinose sugar. The revertants of mutant strain showed two different suppressor mutation R1 and Su^B. R1is the original mutation in L-ribulokinase and not able to metabolize arabinose sugar as the enzyme encoded by the wild type allele. The second suppressor mutation Su^B is not linked to the original mutation. However, the second suppressor able to produce L-ribulokinase in conjunction with R1 mutation

b.

Summary Introduction

To explain:

The way in which R1 and Su^B restore the ability of araB14 to metabolize arabinose and why Su^B is able to more fully restore this ability.

Expert Solution & Answer

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Answer 3

Question

Chapter 18.1, Problem 45CQ

a.

Summary Introduction

To determine:

R1 and Su^B mutations are intragenic suppressors or intergenic suppressors.

Introduction:

Person B and Person C conducted nonsense mutation on E. coli strain. During mutation, the structural gene encodes an enzyme called L-ribulokinase that allows the bacteria to metabolize arabinose sugar. The revertants of mutant strain showed two different suppressor mutation R1 and Su^B. R1is the original mutation in L-ribulokinase and not able to metabolize arabinose sugar as the enzyme encoded by the wild type allele. The second suppressor mutation Su^B is not linked to the original mutation. However, the second suppressor able to produce L-ribulokinase in conjunction with R1 mutation

b.

Summary Introduction

To explain:

The way in which R1 and Su^B restore the ability of araB14 to metabolize arabinose and why Su^B is able to more fully restore this ability.

Expert Solution & Answer

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Answer 4

Question

Chapter 18.1, Problem 45CQ

a.

Summary Introduction

To determine:

R1 and Su^B mutations are intragenic suppressors or intergenic suppressors.

Introduction:

Person B and Person C conducted nonsense mutation on E. coli strain. During mutation, the structural gene encodes an enzyme called L-ribulokinase that allows the bacteria to metabolize arabinose sugar. The revertants of mutant strain showed two different suppressor mutation R1 and Su^B. R1is the original mutation in L-ribulokinase and not able to metabolize arabinose sugar as the enzyme encoded by the wild type allele. The second suppressor mutation Su^B is not linked to the original mutation. However, the second suppressor able to produce L-ribulokinase in conjunction with R1 mutation

b.

Summary Introduction

To explain:

The way in which R1 and Su^B restore the ability of araB14 to metabolize arabinose and why Su^B is able to more fully restore this ability.

Expert Solution & Answer

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See solution

Answer 5

Chapter 18.1, Problem 45CQ

a.

Summary Introduction

To determine:

R1 and Su^B mutations are intragenic suppressors or intergenic suppressors.

Introduction:

Person B and Person C conducted nonsense mutation on E. coli strain. During mutation, the structural gene encodes an enzyme called L-ribulokinase that allows the bacteria to metabolize arabinose sugar. The revertants of mutant strain showed two different suppressor mutation R1 and Su^B. R1is the original mutation in L-ribulokinase and not able to metabolize arabinose sugar as the enzyme encoded by the wild type allele. The second suppressor mutation Su^B is not linked to the original mutation. However, the second suppressor able to produce L-ribulokinase in conjunction with R1 mutation

b.

Summary Introduction

To explain:

The way in which R1 and Su^B restore the ability of araB14 to metabolize arabinose and why Su^B is able to more fully restore this ability.

Answer 6

Chapter 18.1, Problem 45CQ

a.

Summary Introduction

To determine:

R1 and Su^B mutations are intragenic suppressors or intergenic suppressors.

Introduction:

Person B and Person C conducted nonsense mutation on E. coli strain. During mutation, the structural gene encodes an enzyme called L-ribulokinase that allows the bacteria to metabolize arabinose sugar. The revertants of mutant strain showed two different suppressor mutation R1 and Su^B. R1is the original mutation in L-ribulokinase and not able to metabolize arabinose sugar as the enzyme encoded by the wild type allele. The second suppressor mutation Su^B is not linked to the original mutation. However, the second suppressor able to produce L-ribulokinase in conjunction with R1 mutation

Answer 7

Chapter 18.1, Problem 45CQ

a.

Summary Introduction

To determine:

R1 and Su^B mutations are intragenic suppressors or intergenic suppressors.

Introduction:

Person B and Person C conducted nonsense mutation on E. coli strain. During mutation, the structural gene encodes an enzyme called L-ribulokinase that allows the bacteria to metabolize arabinose sugar. The revertants of mutant strain showed two different suppressor mutation R1 and Su^B. R1is the original mutation in L-ribulokinase and not able to metabolize arabinose sugar as the enzyme encoded by the wild type allele. The second suppressor mutation Su^B is not linked to the original mutation. However, the second suppressor able to produce L-ribulokinase in conjunction with R1 mutation

Answer 8

b.

Summary Introduction

To explain:

The way in which R1 and Su^B restore the ability of araB14 to metabolize arabinose and why Su^B is able to more fully restore this ability.

Answer 9

b.

Summary Introduction

To explain:

The way in which R1 and Su^B restore the ability of araB14 to metabolize arabinose and why Su^B is able to more fully restore this ability.

Answer 10

Expert Solution & Answer

Answer 11

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Answer 12

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Answer 13

Ch. 18.1 - Prob. 17AQP Ch. 18.1 - Prob. 18AQP Ch. 18.1 - Prob. 19AQP Ch. 18.1 - Prob. 1CC Ch. 18.1 - Prob. 1COQ Ch. 18.1 - Prob. 20AQP Ch. 18.1 - Prob. 21AQP Ch. 18.1 - Prob. 22AQP Ch. 18.1 - Prob. 23AQP Ch. 18.1 - Prob. 24AQP

To explain: The way in which R1 and SuB restore the ability of araB14 to metabolize arabinose and why SuB is able to more fully restore this ability.

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Chapter 18 Solutions

To explain:

The way in which R1 and Su^B restore the ability of araB14 to metabolize arabinose and why Su^B is able to more fully restore this ability.