EBK INQUIRY TO LIFE - ACCESS CARD
EBK INQUIRY TO LIFE - ACCESS CARD
16th Edition
ISBN: 9781260918403
Author: Mader
Publisher: INTER MCG
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Chapter 13, Problem 1TC

Some organisms, such as invertebrates, have no lymphocytes and thus lack an adaptive system, but they have some components of an innate immune system, including phagocytes and certain protective proteins. What are some general features of innate immunity that make it very valuable to organisms lacking more specific antibody- and cell-mediated responses? What are some disadvantages to having only an innate immune system?

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What distinguishes constitutive resistance from an induced response?  What is the difference between innate and acquired immunity?  What evidence is there that acquired immunity exists in organisms other than vertebrates.
Which of the following describes the self-tolerance of adaptive immunity?   A)  a given immune response acts against a specific antigen possessed by a pathogen B)  both cells and antibodies are involved in immune responses C)  immune responses against previously encountered pathogens are faster and more potent D)  immune responses against the body's own molecules do not normally occur E)  pathogens stimulate cells involved in adaptive immunity
The classical complement pathway is initiated by C1q binding to the surface of a pathogen. In some cases, C1q can directly bind the pathogen, for instance by recognizing proteins of bacterial cell walls, but in most cases C1q binds to IgM antibodies that are bound to the pathogen surface. How does this IgM-binding feature of C1q contribute to rapid, innate immune responses rather than to slow, adaptive responses? C1q induces B lymphocytes to begin secreting antibody within hours of pathogen exposure. Natural antibody that binds to many microbial pathogens is produced prior to pathogen exposure. C1q binds to C-reactive protein which then binds to IgM on the pathogen surface. C1q directly induces inflammation, recruiting phagocytes and antibodies from the blood into the infected tissue. C1q binds to dendritic cells in the infected tissue, inducing them to secrete inflammatory cytokines.

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EBK INQUIRY TO LIFE - ACCESS CARD

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