You're taking a train from Istanbul to Zagreb, and there has been a murder! Fortunately, a world-class detective is on the train with you and the train's owner ask him take over the case. You are not a suspect, but you are an expert in human physiology. You examine the body for "rigor mortis" to estimate the time of death. Rigor mortis occurs because, at death, all the existing ATP is rapidly depleted and no new ATP is made. The lack of ATP prevents the cross-bridge cycle, and the muscles become extremely stiff. You explain all of this to the detective, Hercule P, but he's confused why the lack of ATP would make the body stiff rather than limp. After all, isn't the primary function of sarcomeres to build tension in muscles? You tell Hercule that the lack of ATP actually prevents:
You're taking a train from Istanbul to Zagreb, and there has been a murder! Fortunately, a world-class detective is on the train with you and the train's owner ask him take over the case. You are not a suspect, but you are an expert in human physiology. You examine the body for "rigor mortis" to estimate the time of death. Rigor mortis occurs because, at death, all the existing ATP is rapidly depleted and no new ATP is made. The lack of ATP prevents the cross-bridge cycle, and the muscles become extremely stiff. You explain all of this to the detective, Hercule P, but he's confused why the lack of ATP would make the body stiff rather than limp. After all, isn't the primary function of sarcomeres to build tension in muscles? You tell Hercule that the lack of ATP actually prevents:
Human Anatomy & Physiology (11th Edition)
11th Edition
ISBN:9780134580999
Author:Elaine N. Marieb, Katja N. Hoehn
Publisher:Elaine N. Marieb, Katja N. Hoehn
Chapter1: The Human Body: An Orientation
Section: Chapter Questions
Problem 1RQ: The correct sequence of levels forming the structural hierarchy is A. (a) organ, organ system,...
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Hi, May I get help with this question for physiology? An explanation leading to the correct answer would be helpful!
Scenario: You're taking a train from Istanbul to Zagreb, and there has been a murder! Fortunately, a world-class detective is on the train with you and the train's owner ask him take over the case. You are not a suspect, but you are an expert in human physiology. You examine the body for "rigor mortis" to estimate the time of death. Rigor mortis occurs because, at death, all the existing ATP is rapidly depleted and no new ATP is made. The lack of ATP prevents the cross-bridge cycle, and the muscles become extremely stiff. You explain all of this to the detective, Hercule P, but he's confused why the lack of ATP would make the body stiff rather than limp. After all, isn't the primary function of sarcomeres to build tension in muscles?
You tell Hercule that the lack of ATP actually prevents:
![**Muscle Contraction Mechanisms: Key Terms**
1. **The closing of CA2+ leak channels on the sarcoplasmic reticulum:**
- A critical process to stop calcium ions from continuously leaking into the muscle cell, thus helping to regulate muscle contraction and relaxation.
2. **The troponin & tropomyosin from blocking the actin on the thin filament:**
- Troponin and tropomyosin are proteins that regulate the binding of myosin to actin in muscle cells. Their repositioning allows muscle contraction to proceed.
3. **The decoupling of the cross bridges from the actin:**
- Cross bridges, formed by myosin heads attaching to actin, detach to allow the muscle fibers to reset for the next contraction cycle.
4. **YES:**
- A placeholder or confirmation response, possibly related to user interaction or confirmation in an educational setting.
5. **The power stroke:**
- The action by which myosin heads pivot, pulling the actin filaments toward the center of the sarcomere, ultimately leading to muscle contraction.](/v2/_next/image?url=https%3A%2F%2Fcontent.bartleby.com%2Fqna-images%2Fquestion%2F25d5c273-ce26-424b-bd36-a8a6b930ab8d%2F851e53ac-4c80-4b9a-a919-0f8a675ae418%2Fg20td2_processed.jpeg&w=3840&q=75)
Transcribed Image Text:**Muscle Contraction Mechanisms: Key Terms**
1. **The closing of CA2+ leak channels on the sarcoplasmic reticulum:**
- A critical process to stop calcium ions from continuously leaking into the muscle cell, thus helping to regulate muscle contraction and relaxation.
2. **The troponin & tropomyosin from blocking the actin on the thin filament:**
- Troponin and tropomyosin are proteins that regulate the binding of myosin to actin in muscle cells. Their repositioning allows muscle contraction to proceed.
3. **The decoupling of the cross bridges from the actin:**
- Cross bridges, formed by myosin heads attaching to actin, detach to allow the muscle fibers to reset for the next contraction cycle.
4. **YES:**
- A placeholder or confirmation response, possibly related to user interaction or confirmation in an educational setting.
5. **The power stroke:**
- The action by which myosin heads pivot, pulling the actin filaments toward the center of the sarcomere, ultimately leading to muscle contraction.
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