Which of the following best explains why vaccination of humans against rabies can be delayed until after exposure to a potential source of infection? A) Antibody responses are more rapid than those against other viruses B) T lymphocyte responses are more rapid than those against other viruses C) The virus does not infect any cells for several weeks or months D) The virus does not reach i target organ for several weeks or months

Which of the following best explains why vaccination of humans against rabies can be delayed until after exposure to a potential source of infection? A) Antibody responses are more rapid than those against other viruses B) T lymphocyte responses are more rapid than those against other viruses C) The virus does not infect any cells for several weeks or months D) The virus does not reach i target organ for several weeks or months

Concepts of Biology

1st Edition

ISBN:9781938168116

Author:Samantha Fowler, Rebecca Roush, James Wise

Publisher:Samantha Fowler, Rebecca Roush, James Wise

Chapter17: The Immune System And Disease

Section: Chapter Questions

Problem 16CTQ: Why can’t dogs catch the measles?

See similar textbooks

Similar questions

Why is immunization after being bitten by a rabid animal so effective?
Arrange the following list into the correct sequence for part of the cycle of a retrovirus: 1. dsDNA integrated into host DNA 2.viral proteins synthesized on host ribosomes 3. viral DNA uses host enzymes to transcribe viral RNA 4. reverse transcriptase catalyzes synthesis of ssDNA 5. synthesis of second DNA strand (a) 5, 2, 1, 3, 4 (b) 5, 2, 3, 4, 1 (c) 4, 5, 1, 3, 2 (d) 4, 1, 2, 3, 5 (e) 2, 1, 3, 4, 5
There have been recurring cases of mad-cow disease in the United Kingdom since the mid-1990s. Mad-cow disease is caused by a prion, an infectious particle that consists only of protein. In 1986, the media began reporting that cows all over England were dying from a mysterious disease. Initially, there was little interest in determining whether humans could be affected. For 10 years, the British government maintained that this unusual disease could not be transmitted to humans. However, in March 1996, the government did an about-face and announced that bovine spongiform encephalopathy (BSE), commonly known as mad-cow disease, can be transmitted to humans, where it is known as variant Creutzfeldt-Jakob disease (vCJD). As in cows, this disease eats away at the nervous system, destroying the brain and essentially turning it into a spongelike structure filled with holes. Victims experience dementia; confusion; loss of speech, sight, and hearing; convulsions; coma; and finally death. Prion diseases are always fatal, and there is no treatment. Precautionary measures taken in Britain to prevent this disease in humans may have begun too late. Many of the victims contracted it over a decade earlier, when the BSE epidemic began, and the incubation period is long (vCJD has an incubation period of 10 to 40 years). A recent study concluded that 1 in 2,000 people in Great Britain carry the abnormally folded protein that causes vCJD. In spite of these numbers, the death rate from vCJD remains low. It is not clear whether this means that the incubation period for the disease is much longer than previously thought, or whether they may never develop the disease. What measures have been taken to stop BSE?
There have been recurring cases of mad-cow disease in the United Kingdom since the mid-1990s. Mad-cow disease is caused by a prion, an infectious particle that consists only of protein. In 1986, the media began reporting that cows all over England were dying from a mysterious disease. Initially, there was little interest in determining whether humans could be affected. For 10 years, the British government maintained that this unusual disease could not be transmitted to humans. However, in March 1996, the government did an about-face and announced that bovine spongiform encephalopathy (BSE), commonly known as mad-cow disease, can be transmitted to humans, where it is known as variant Creutzfeldt-Jakob disease (vCJD). As in cows, this disease eats away at the nervous system, destroying the brain and essentially turning it into a spongelike structure filled with holes. Victims experience dementia; confusion; loss of speech, sight, and hearing; convulsions; coma; and finally death. Prion diseases are always fatal, and there is no treatment. Precautionary measures taken in Britain to prevent this disease in humans may have begun too late. Many of the victims contracted it over a decade earlier, when the BSE epidemic began, and the incubation period is long (vCJD has an incubation period of 10 to 40 years). A recent study concluded that 1 in 2,000 people in Great Britain carry the abnormally folded protein that causes vCJD. In spite of these numbers, the death rate from vCJD remains low. It is not clear whether this means that the incubation period for the disease is much longer than previously thought, or whether they may never develop the disease. If you were traveling in Europe, would you eat beef? Give sound reasons why or why not.
The viral ________ plays a role in attaching a virion to the host cell. a. core b. capsid C. envelope d. both b and c
Figure 17.5 Which of the following statements about virus structure is true? a. All viruses are encased in a viral membrane. b. The capsomere is made up of small protein subunits called capsids. c. DNA is the genetic material in all viruses. d. Glycoproteins help the virus attach to the host cell.
The man pictured in Figure 4.17 wears skin ornaments applied by piercing. Among other functions, the skin is a barrier to potentially dangerous bacteria, and some people object to extensive body piercing on the grounds that it opens the door to infections. Explain why you do or dont agree with this objection. Figure 4.17 This young man has chosen to undergo multiple body piercings.
New research suggests a link between some microbes that normally live in the body and seemingly unrelated major illnesses. The gum disease called periodontitis itself is not life-threatening, for instance, but it is a fairly good predictor for heart attacks. Bacteria that cause gum disease can trigger inflammation. Thinking back to your reading in Chapter 7, how do you suppose that this response also may be harmful to the heart?
Figure 17.6 Influenza virus is packaged in a viral envelope, which fuses with the plasma membrane. This way, the virus can exit the host cell without killing it. What advantage does the virus gain by keeping the host cell alive?
The genome of a virus consists of (a) DNA (b) RNA (c) prions (d) DNA and RNA (e) DNA or RNA
______ are barriers to pathogens at body surfaces. a. Intact skin and mucous membranes b. Tears, saliva, and gastric fluid c. Resident bacteria d. All are correct