the experiment of the impacts of sedative and hypnotic acivities in MEJP leaves, the independent variables are the different behavioral models while the amount of dosage is the dependent variables, what can be the cofounding variables and the intervening variables
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In the experiment of the impacts of sedative and hypnotic acivities in MEJP leaves, the independent variables are the different behavioral models while the amount of dosage is the dependent variables, what can be the cofounding variables and the intervening variables?
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- In the experiment of the impacts of sedative and hypnotic acivities in MEJP leaves, does the different behavioral models in mice are the independent variables while the amount of dosage is the dependent variables?What is the intent behind testing simple main effects on a significant interaction?when you should ignore the main effects and just focus on the simple main effects? How to to plot cell means on a graph? Please provide brief explanations. For thumbs up. Thank you.Thank you so much, The second question is: Analysis (Analyzing Cues) Which client conditions are consistent with the cues? Do the cues support a particular client condition? What cues are a cause for concern? What other information would help to establish the significance of a cue? Thank you!!
- Part 2: Please read the following scenarios. For each scenario, identify the confound and suggest how it might be removed in a follow-up experiment. McCann and Holmes (1984) tested the hypothesis that exercise reduces depression. They randomly assigned depressed undergraduate women either to an exercise condition (attending an aerobics class a couple of times a week for ten weeks) or to a relaxation training condition (the individuals relaxed at home by watching a videotape over the same period of time). Although the results showed that the exercise group reported less depression at the end of the ten-week period than did the relaxation group, why can't the researchers conclude that exercise reduces depression?how to write a tactful and diplomatic critique on this section? Despite overlaps in clinical symptomatology, autism and ADHD may be associated with opposite autonomic arousal profiles which might partly explain altered cognitive and global functioning. We investigated autonomic arousal in 106 children/adolescents with autism, ADHD, co-occurring autism/ADHD, and neurotypical controls. Heart rate variability was recorded during resting-state, a ‘passive’ auditory oddball task and an ‘active’ response conflict task. Autistic children showed hyper-arousal during the active task, while those with ADHD showed hypo-arousal during resting-state and the passive task. Irrespective of diagnosis, children characterised by hyper-arousal showed more severe autistic symptomatology, increased anxiety and reduced global functioning than those displaying hypo-arousal, suggesting the importance of considering individual autonomic arousal profiles for differential diagnosis of autism/ADHD and when…Scenerio: Is Cognitive Processing Therapy (CPT) more effective in treating PTSD than Prolonged Exposure therapy (PE)? Question 1: Does this question represent a correlational or experimental research design? Why? Question 2: Which of the two models would state that there is nothing interesting going on between CPT and PE in treating PTSD? Question 3: . State the alternative hypothesis for this study. Question 4: State the null hypothesis for this study. Question 5: Are your hypotheses directional or non-directional? How do you know this? Question 6: Suppose you run the study, and the results come out with a p value of .23. What can you conclude about the difference between the treatments? Question 7: In this case, would you decide to reject or fail to reject the null hypothesis? Why?
- What role does associative learning play in drug relapse? Be sure to include memory engrams and synaptic plasticity and explain this in a few sentences.Kirsch (2010) argues that in double-blind clinical trials to test the effects of antidepressants, a large fraction of patients figure out whether they have been given the antidepressant or the placebo by noticing the presence or absence of known side effects of the antidepressant. Doctors evaluating the patients are also able to determine which treatment patients are receiving. How might this situation affect the results of the clinical trial? Specifically, is the treatment effect (difference between the means of the antidepressant and placebo treatments) likely to be overestimated, underestimated, or unaffected by this knowledge?40. Use the diagram below for this question. In this research study, Jager et al. (Frontiers Psychiatry, 2019) used the elevated-plus maze test in mice to examine the effects of increasing doses of the psychostimulant known as methylphenidate (MPH). From the pictures and graph provided, what could they conclude? Elevated-plus maze test after acute treatment with methylphenidate (MPH) Time in Open Arms Venice Contra MPH 3mghg MPH 10 mg a. MPH significantly augmented anxiety in a dose-dependent manner. b. MPH significantly reduced anxiety in a dose-dependent manner. c. MPH significantly augmented depression in a dose-dependent manner. d. MPH significantly reduced depression in a dose-dependent manner. e. None of the above make sense.. % TotalTime Mine
- in table 4 we see the p value is 0.053 Detecting a tendency in the correlation between NCI and stress. And when looking at figure 3 we can see that correlation between NCI and stress? why is that explain how the graph shows that?In homeless individuals with opioid addiction (P), does the implementation of supervised injection sites (I), as compared to abstinence-promotion programs (C), lead to a more significant reduction in substance-related self-harm incidents (O) over five years (T)? This is my PICOT research question though due to contemporary nature of this topic, I am unable to find primary research articles in the databases to use for a literature Review. How can I reword or fix this PICOT question to retain more articles.A researcher wishes to determine if vitamin E supplements could increase cognitive ability among elderly women. In 1999 the researcher recruits a sample of elderly women age 75-80. At the time of the enrollment into the study, the women were randomized to either take Vitamin E, or a placebo for six months. At the end of the six month period, the women were given a cognition test. Higher scores on this test indicate better cognition. The mean and standard deviation of the test scores of 81 women who took vitamin E supplements was 27 and 6.9 respectively. The mean and standard deviation of the test scores of the 90 women who took placebo supplements was 24 and 6.2, respectively.C Computea 95% confidence interval for the mean difference in cognition test scores between Vitamin E and placebo group.