The diagrams below represent nucleic acid genomic material and a finished product afterthe viral polymerase acted on the genomic material. Name the virus (either family, genus,or particular virus is acceptable) that these diagrams represent.A isB isC isDisGenomic materialProduct+ sense RNAA 5' VPB-sense RNA3'VPg 5'-sense RNA5' capDerived from infected cellB3'5'AAAAA(A) 3+ sense RNAC+ sense RNA-sense RNA+sense RNA5'3'

let's break down the reasoning behind identifying the viruses based on the provided diagrams and…

Answered: The diagrams below represent nucleic…

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O O O A new virus is discovered. Its RNA genome encodes several proteins, including a reverse transcriptase enzyme. What can you conclude about replication of this virus? Its replication involves a double-stranded RNA intermediate Its replication involves a DNA copy of its genome Its replication depends entirely on cellular enzymes Its replication is entirely independent of cellular enzymes 00 A deficiency in Base Excision Repair (BER) would primarily sensitize you to mutations caused by radiation, X-rays O Chemicals in smoke, and UV radiation O Unavoidable, spontaneously occurring DNA damage
The illustration best describes: A dormant bacterial infection A dormant viral infection An active bacterial infection An active viral infection
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Berkely’s experiment with temperature sensitive viral mutant showed that ----. virus could not make the viral protein at high temperature viral genome remained in the cell after the initial infection continuous presence of viral protein was required to maintain the transformed malignant phenotype of cells. all of the above a and b please answer it quickly
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Can someone help me with my virology homework? Can you explain how to interpret these three figures?
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answer doesn't have to be long i just need an explanation, thank you! The whole world right now is concerned (or they should be) about the appearance of new variants of SARS-CoV-2. Among other things, these variant viruses have mutations that change the amino acid sequence of the spike protein on the virus surface. While this is concerning, I would like to propose that there is a limit to how far these mutations can change the spike protein. Why can't there be an endless number of new mutations of this viral protein? How would these amino acid changes affect how the virus works?