explain the picture in full and detail about steroidogenesis in the ovaries (two cells theory) GranulosaТесаMitocondriaP450sccPregnenolonaEstradiolSIARLiquidofolicularATP173-HSD-1ColesterolCAMPATPSF-1EstronaSF-1CAMP33-HSD-IIP450c17P450aromGgFSHFSH-RAndrostenedionaAndrostenedionaLH-RLHCitosolAGranulosaLDLТеcаMitocondriaMitocondriaP450sccP450scePregnenolonaColesterolPregnenolonaSARLHLH-RSIARColesteral33-HSD-IIATPSF-1ProgesteronaSF-1CAMPAATP33-HSD-IIEstradiol(G.lP450c17CAMP173-HSD-1EstronaAndrostenedionaLH-RLHP450aromAndrostenediona ACitosolFSHFSH-RCitosolBCiclasaCidasaMembrana basal> CirculaciónCirculaciónCiclasaCiclasawwww E

The production of steroids is known as "steroidogenesis". The steroid is a molecule with the…

Granulosa Теса Mitocondria P450scc Pregnenolonia Estradiol SIAR Liquido folicular ATP 178-HSD-1 Colesterol CAMP ATP SF-1 Estrona Gg SF-1 CAMPA 35-HSD-II P450c17 P450arom FSH FSH-R Androstenediona Androstenediona LH-R LH Citosol A Granulosa Теса LDL Mitocondria Mitocondria P450sce P450sce Pregnenolona Colesterol SIAR Pregnenolona LH LH-R SIAR Colesterol 33-HSD-I ATP SF-1 Progesterona SF-1 CAMPA ATP 3i-HSD-II Estradiol P450c17 CAMP 17-HSD-1 Estrona Androstenediona LH-R LH P450arom AndrostenedionaA Citosol FSH FSH-R Citosol Ciclasa Cidasa Membrana basal > Circulación Circulación Ciclasa Ciclasa

Granulosa Теса Mitocondria P450scc Pregnenolonia Estradiol SIAR Liquido folicular ATP 178-HSD-1 Colesterol CAMP ATP SF-1 Estrona Gg SF-1 CAMPA 35-HSD-II P450c17 P450arom FSH FSH-R Androstenediona Androstenediona LH-R LH Citosol A Granulosa Теса LDL Mitocondria Mitocondria P450sce P450sce Pregnenolona Colesterol SIAR Pregnenolona LH LH-R SIAR Colesterol 33-HSD-I ATP SF-1 Progesterona SF-1 CAMPA ATP 3i-HSD-II Estradiol P450c17 CAMP 17-HSD-1 Estrona Androstenediona LH-R LH P450arom AndrostenedionaA Citosol FSH FSH-R Citosol Ciclasa Cidasa Membrana basal > Circulación Circulación Ciclasa Ciclasa

Human Anatomy & Physiology (11th Edition)

11th Edition

ISBN:9780134580999

Author:Elaine N. Marieb, Katja N. Hoehn

Publisher:Elaine N. Marieb, Katja N. Hoehn

Chapter1: The Human Body: An Orientation

Section: Chapter Questions

Problem 1RQ: The correct sequence of levels forming the structural hierarchy is A. (a) organ, organ system,...

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Hypogonadism is a condition where the body does not produce enough testosterone. The symptoms of hypogonadism include infertility and a decrease in secondary sex characteristics. There are two types of hypogonadism. Primary hypogonadism is caused by problems stemming from the testicles, and secondary hypogonadism is caused by problems stemming from the hypothalamus or the pituitary gland. Individuals with hypogonadism are often treated with testosterone replacement therapy. What would be the result of testosterone injections in individuals with hypogonadism? Select one: a. GnRH LH Testosterone Decrease Decrease Increase b. GnRH LH Testosterone Decrease Decrease Decrease c. GnRH LH Testosterone Increase Increase Increase d. GnRH LH Testosterone Increase Increase Decrease
A B C D Hypothalamus Pituitary glands Ovaries Uterus Hormonal Nervous Dilation Placental Expulsion Positive Negative Use the table above to complete the following sentence. Oxytocin, an important hormone involved in parturition, is produced by the ____A____ and released through ____B____ stimulation. Oxytocin stimulates uterine contractions which in turn stimulate the release of prostaglandins. Both oxytocin and prostaglandins are released during the ____C____ stage of labour through a ______D____ feedback loop. A:AnswerB:AnswerC:AnswerD:Answer
R4
Discuss how the concept of cell-to-cell communication applies to the endocrine svstem
Gonadocorticoid A. synthesized by the adrenal medulla are primarily androgens. B. Production by the adrenal gland is insignificant as compared to sex hormone release from the gonads during late puberty C. Inhibition (of secretion) is highly dependent on a negative feedback loop involving ACTH D. Hypersecretion can result in androgenital syndrome also called feminization E. All of these are correct
DRAW IT In mammals, milk production by mammary glandsis controlled by prolactin and prolactin-releasing hormone.Draw a simple sketch of this pathway, including glands, tissues,hormones, routes for hormone movement, and effects.
Disorder of Sex Determination Now that you have reviewed typical sexual determination in humans, it is time to review some case studies of patients that present with sex determination disorders. Using the symptoms presented, research online to determine the nature of the disorder, its mechanism (what causes the disorder) and also any complications and treatment options that may exist. Case 1 A 16-year old patient presents with the following symptoms: Presents as normal external female Did not develop breasts at puberty Did not proceed through menarche (first menstrual cycle) Genetic karyotyping revealed XY Gonadal 'streaks' found upon internal examination Diagnosis Mechanism Possible Complications and Recommendations
provide a summarization of the following paragraph(s) in bullet points Perimenopausal and Postmenopausal Years Female aging is associated with genetically determineddeclining DHEAS and T (Fig. 2) (9, 10, 15, 16). In parallel, sexual desire declines with age (17, 18). Decreased sexual desire with ‘‘reverse adrenarche’’has long been associated with clinical disruptions (Table 2) of endogenous androgen decline. Examples include oophorectomy, oral estrogen therapy, adrenal insufficiency, corticosteroid adrenal suppression, and hypopituitarism (19–21). Aging afflicts sexual endocrinology in other ways. Engagement in a secure and attractive relationship, a safe place, good health, and no drugs remain as core issues. Declining estrogens. Estradiol secretion, chaotic during peri- menopausal years, declines to very low levels after menopause. Estrogen withdrawal increases tissue fragility, increases vaginal and urinary infections, irritation, dryness, urogenital pain, and susceptibility to…
Recall what you learned in class about the experiments that examined the effect of early treatment with hormones on adult reproductive behavior. Match the following behavioral outcomes with each of the manipulations in the questions below. Each answer may be used more than once. An XY animal is castrated (gonadectomized) at birth and treated with testosterone the same day and the next day. What reproductive behavior, if any will this animal show when treated with testosterone as an adult? An XY animal is castrated (gonadectomized) at birth and treated with estradiol the same day and the next day. What reproductive behavior, if any will this animal show when treated with estradiol and progesterone as an adult? An XX animal is not surgically manipulated, but is treated with testosterone on the day of its birth and the day…
Place labels in their respective targets
. A young male patient is currently undergoing therapy using an androgen. What life span considerations must you consider?
Which of the following is the false statement? O All pituitary hormones, including thyroid hormones, are affected by loss of reproductive function O FDA does not approve use of individually compounded bioidentical hormones for treatment of perimenopausal symptoms due to lack of standardization and demonstrated effectiveness After menopause, the vagina becomes more susceptible to infection due to decreases in lactobacilli O The risk of coronary heart disease increases in postmenopausal women and becomes the number one overall killer of women