answer question in image **Question:**Acetylcholine released into the junction between a motor neuron and a skeletal muscle binds to a ligand-gated sodium (Na+) channel and opens it, allowing sodium (Na+) to enter the muscle cell. What effect would this have on the likelihood that the skeletal muscle will fire an action potential relative to the scenario with no acetylcholine?**Options:**- A. More likely to fire an action potential- B. Less likely to fire an action potential- C. No effect

The neuromuscular junction (NMJ) is a specialized synapse that allows motor neurons to communicate with muscle fibers. When an action potential reaches the presynaptic terminal of a motor neuron, it triggers the release of neurotransmitter acetylcholine (ACh) into the synaptic cleft. ACh then binds to receptors on the postsynaptic membrane of the muscle fiber, causing a depolarization of the membrane. The answer is: A. More likely to fire an action potentialWhen acetylcholine binds to the ligand-gated sodium channel, it opens the channel and allows sodium ions to enter the muscle cell. This influx of sodium ions depolarizes the muscle cell, making it more likely to fire an action potential.The sequence of process thst happens is:Acetylcholine is released from the motor neuron terminal.Acetylcholine binds to the ligand-gated sodium channel on the muscle cell.The sodium channel opens, allowing sodium ions to enter the muscle cell.The influx of sodium ions depolarizes the muscle cell.If the depolarization is sufficient, an action potential is fired.Therefore, the answer is that the release of acetylcholine into the neuromuscular junction makes the skeletal muscle more likely to fire an action potential.The answer is: A. More likely to fire an action potential