MSN 571 SEC2 WEEK 3 DQ1.edited (1)

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Dec 6, 2023

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MSN 571 SEC2 WEEK 3 DQ1 (IN PROGRESS) Question 1 With the 2033 Joint National Committee's seventh report (JNC 7) turning obsolete and the 2011 Institute of Medicine report requesting a better, evidence-based guiding principle. 7, 8, (the Right Joint National Committee (JNC 8) was at first selected to develop an up-to-date treatment guideline for high blood pressure (HTN) under the indications of the National Institutes of Health (NIH). The NIH eventually withdrew from the regulation development practice at the end of the JNC 8’s development; nevertheless, the panel determined to make public their proposals autonomously. Worldwide, hypertension (HTN) is the leading risk factor for cardiovascular disease (CVD) mortality and morbidity. In this Nation, hypertension is placed first among modifiable risk factors in the populace due to CVD risk, accountable for the most significant part of coronary heart disease (CHD), heart failure (HF), and stroke incidents. In adults with HTN, the control of HTP with antihypertensive treatment reduces the chances of CVD and mortality from all causes (Carey et al., 2021). The panel members selected for the Eight Joint National Committee (JNC-8) revealed a report proposing a higher BP goal SBP/DBP< 150/90 mm Hg for adults > 60 years in comparison with the 2003 JNC-7 recommended target SBP/DBP < 140/90 MM Hg. A small group report from five out of 17 JNC-8 panel participants informed that relaxation of the blood pressure target could decrease the amount of antihypertensive medication and the level of control of blood pressure (Carey et al., 2021). The panel members selected for the Eight Joint National Committee (JNC-8) revealed a report proposing a higher BP goal SBP/DBP< 150/90 mm Hg for adults > 60 years in comparison with the 2003 JNC-7 recommended target SBP/DBP < 140/90 MM Hg. A small group report
from five out of 17 JNC-8 panel participants informed that relaxation of the blood pressure target could decrease the amount of antihypertensive medication and the level of control of blood pressure (Carey et al., 2021). ACC/AHA recommendations on the Primary Prevention of Cardiovascular Disease are that all clients with hypertension. In 2017, the American College of Cardiology (ACC) and the American Heart Association (AHA) defined HTN in stages (Khalil & Zeltser, 2023). 1. Normal blood pressure as systolic blood pressure is less < 120 mm Hg and diastolic blood pressure is < 80 mm Hg. 2. Elevated blood pressure as systolic blood pressure is 120 mm Hg to 130 mm Hg, and diastolic blood pressure is less < 80 mm Hg. 3. Stage 1 HTN is systolic blood pressure 130 mm Hg to 139 mm Hg or diastolic blood pressure 80 mm Hg to 89 mm Hg. 4. Stage 2 HTN is systolic blood pressure of at least 140 mm Hg or diastolic at least 90 mm Hg. 5. Hypertensive crises are systolic blood pressure over 180 mm Hg and diastolic blood pressure over 120 mm Hg. Question 2 For African-origin patients who do not have a history of chronic kidney disease (CKD), the treatment must start with thiazides and CCBs. Norvasc is a marketing name, and amlodipine is a generic name. This medication is classified as a calcium channel blocker; this medication dose should start in Adults with 5 mg once a day for the first week. The dose is established in each individual and can be increased once at least one to two weeks of use. The highest dose is 10 mg daily. For the elderly, the dose for the first week is 2.5 mg once daily for the first week too. Microzide is the commercial name for Sodium hydrochlorothiazide, a generic name. The medicine class is thiazides, and the initial dose recommended is 12.5 mg daily, gradually increasing up to 50 mg daily as needed. It can be taken by itself or in combination with other
hypertensive medication. The dose is adapted to the specific needs of the patient and could be titrated in reaction to their illness and condition. The highest dose is 100 mg daily. For patients 65 and older, initially 12.5 daily, it can be titrated as needed until the desired effect is reached Question 3 Hydrochlorothiazide inhibits sodium chloride reabsorption in the distal tubule. It results in more Na excretion in the kidney with fluid. The pharmacological effect starts 2 hours after the oral has been taken and peaks in four hours; it lasts approximately six to 12 hours. This mentioned medication is not metabolized, and most is excreted in the urine unaffected. It causes hypokalemia and loss of bicarbonate (Herman & Bashir, 2023). The long-term actions of HCTZ once decreasing blood pressure have yet to be entirely understood. When HCTZ is given acutely, the medication decreases blood pressure by triggering diuresis and lowering plasma volume. Nevertheless, for chronic use, HCTZ decreases blood pressure by reducing peripheral resistance. It is not clear how the drug causes vasodilation; however, lab evidence implies that it could be inhibiting the enzyme carbonic anhydrase, inactivating the smooth muscle receptors to the rice in Ca, or inhibiting the kidneys' autoregulation (Herman & Bashir, 2023). Amlodipine decreases blood pressure and peripheral vascular resistance by relaxing coronary vascular smooth muscle. It helps increase coronary vasodilation by blocking Ca ion transmembrane participation in vascular and cardiac smooth muscles. Amlodipine blocks the voltage-dependent L-type calcium channels, thus preventing the first influx of calcium. The result of decreased intracellular calcium is decreased vascular smooth muscle contractility, resultant vasodilation, and increased smooth muscle relaxation. Also, amlodipine has enhanced
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vascular endothelial function in patients with HTN. Amlodipine decreases blood pressure when stimulating smooth muscle vasodilation and relaxation (Bulsara & Cassagnol, 2023). The function of amlodipine in relieving stable angina is because of the lowering afterload due to its hypertensive and vasodilatory properties. It decreases afterload, which results in lowering myocardial oxygen demand at exertion level, while the heart does need to work as much to pump blood in the systemic circulation. Amlodipine can relieve Prinzmetal or variant angina, blocking coronary spasms and restoring the coronary arteries’ blood flow (Bulsara & Cassagnol, 2023). Question 4 Possible side effects of hydrochlorothiazide are hyperuricemia, dyslipidemia, dizziness, headaches, hyperglycemia, restlessness, rash, and erectile dysfunctions (Papadaskis et al., 2022). Amlodipine side effects include edema, dizziness, headaches, palpitations, flushing, hypotension, tachycardia, urinary frequency, constipation, and bloating (Papadaskis et al., 2022). Question 5 Hydrochlorothiazide interactions, when taken with alcohol, could increase chances of toxicity of lithium, and when taken with drugs that cause torsades de pointes, like astemizole, digitalis glycoside, sotalol, pimozide, and terfenadine, it can increase the chances of hypokalemia, as well as increasing the risk of orthostatic hypotension once combined with opioids and barbiturates, it can also have an additive effect on first dose hypotension after combined with an ACE inhibitor (Herman & Bashir, 2023). Amlodipine interactions when taking grapefruit or grapefruit juice, the plasma concentration is elevated; when administered St. John’s wort, the plasma concentrations were low. Immunosuppressive medications increase systemic plasma concentration, whereas
simvastatin triggers an increase in serum concentration. Additionally, CYP3A4 enzyme inhibitors generate an increase in exposure. There is a reduction in plasma concentration in the case of CYP3A4 (Bulsara & Cassagnol, 2023). Question 6 Lifestyle changes are recommended for patients with HTN (Papadaskis et al., 2022). Eating fruits and vegetables, low-fat dairy, and low saturated and total fats (DASH diet) has decreased blood pressure. Increase dietary fiber; for 7 g of dietary fiber consumed, cardiovascular risks can decrease by 9% Reduce or stop taking alcohol. Exercise aerobics 30 minutes daily, dynamic 90 to 150 minutes weekly, and isometrics; hand grip four repetitions three times a week. Medication and breathing control References Bulsara, K. G., & Cassagnol, M. (2023). Amlodipine . StartPearls. Bookshelf NCBI. https://www.ncbi.nlm.nih.gov/books/NBK519508/ Carey, R. M., Wright, J. T., Jr, Taler, S. J., & Whelton, P. K. (2021). Guideline-Driven Management of Hypertension: An Evidence-Based Update. Circulation research , 128 (7), 827–846. https://doi.org/10.1161/CIRCRESAHA.121.318083 Herman, L. L., & Bashir, K. (2023). Hydrochlorothiazide. StatPearls. Bookshelf NCBI. https://www.ncbi.nlm.nih.gov/books/NBK430766/ Khalil, H., & Zeltser, R. (2023). Antihypertensive medications. StatPearls. Bookshelf NCBI. https://www.ncbi.nlm.nih.gov/books/NBK554579/
Papadakis, M. A., McPhee, S. T., & Rabow, M.W. (2022). Current medical diagnosis & treatment . McGraw Hill.
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