Biology
12th Edition
ISBN: 9780078024269
Author: Sylvia Mader, Michael Windelspecht
Publisher: McGraw-Hill Education
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Chapter 16, Problem 2TS
You are observing a grouse population in which two feathers
Matings with dark-feathered males: 13 Matings with bright-feathered males: 32 Propose a hypothesis to explain why females apparently prefer bright-feathered males. What selective advantage might there be in choosing a male with alleles that make it more susceptible to predation? What data would help test your hypothesis?
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If you had an unknown microbe, what steps would you take to determine what type of microbe (e.g., fungi, bacteria, virus) it is? Are there particular characteristics you would search for? Explain.
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INTRODUCTION
LABORATORY SIMULATION
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Lab Data
Repeat measurement for tubes 2-5
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Data…
Ch.23
How is Salmonella able to cross from the intestines into the blood?
A. it is so small that it can squeeze between intestinal cells
B. it secretes a toxin that induces its uptake into intestinal epithelial cells
C. it secretes enzymes that create perforations in the intestine
D. it can get into the blood only if the bacteria are deposited directly there, that is, through a puncture
—
Which virus is associated with liver cancer?
A. hepatitis A
B. hepatitis B
C. hepatitis C
D. both hepatitis B and C
—
explain your answer thoroughly
Chapter 16 Solutions
Biology
Ch. 16.1 - 1. List the five conditions necessary for...Ch. 16.1 - Prob. 2CYPCh. 16.2 - Prob. 1CYPCh. 16.2 - Prob. 2CYPCh. 16.2 - Prob. 3CYPCh. 16.3 - Prob. 1CYPCh. 16.3 - 2. Demonstrate how sickle-cell disease is an...Ch. 16 - Prob. 1NS.1QCCh. 16 - Prob. 1NS.2QCCh. 16 - Prob. 1NS.3QC
Ch. 16 - Prob. 1ACh. 16 - Prob. 2ACh. 16 - Prob. 3ACh. 16 - Prob. 4ACh. 16 - Prob. 5ACh. 16 - Prob. 6ACh. 16 - Prob. 7ACh. 16 - Prob. 8ACh. 16 - Prob. 9ACh. 16 - Prob. 10ACh. 16 - Prob. 11ACh. 16 - The continued occurrence of sickle-cell disease...Ch. 16 - Prob. 13ACh. 16 - Prob. 1TSCh. 16 - You are observing a grouse population in which two...Ch. 16 - Prob. 3TSCh. 16 - Prob. 4TS
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- Ch.21 What causes patients infected with the yellow fever virus to turn yellow (jaundice)? A. low blood pressure and anemia B. excess leukocytes C. alteration of skin pigments D. liver damage in final stage of disease — What is the advantage for malarial parasites to grow and replicate in red blood cells? A. able to spread quickly B. able to avoid immune detection C. low oxygen environment for growth D. cooler area of the body for growth — Which microbe does not live part of its lifecycle outside humans? A. Toxoplasma gondii B. Cytomegalovirus C. Francisella tularensis D. Plasmodium falciparum — explain your answer thoroughlyarrow_forwardCh.22 Streptococcus pneumoniae has a capsule to protect it from killing by alveolar macrophages, which kill bacteria by… A. cytokines B. antibodies C. complement D. phagocytosis — What fact about the influenza virus allows the dramatic antigenic shift that generates novel strains? A. very large size B. enveloped C. segmented genome D. over 100 genes — explain your answer thoroughlyarrow_forwardWhat is this?arrow_forward
- Molecular Biology A-C components of the question are corresponding to attached image labeled 1. D component of the question is corresponding to attached image labeled 2. For a eukaryotic mRNA, the sequences is as follows where AUGrepresents the start codon, the yellow is the Kozak sequence and (XXX) just represents any codonfor an amino acid (no stop codons here). G-cap and polyA tail are not shown A. How long is the peptide produced?B. What is the function (a sentence) of the UAA highlighted in blue?C. If the sequence highlighted in blue were changed from UAA to UAG, how would that affecttranslation? D. (1) The sequence highlighted in yellow above is moved to a new position indicated below. Howwould that affect translation? (2) How long would be the protein produced from this new mRNA? Thank youarrow_forwardMolecular Biology Question Explain why the cell doesn’t need 61 tRNAs (one for each codon). Please help. Thank youarrow_forwardMolecular Biology You discover a disease causing mutation (indicated by the arrow) that alters splicing of its mRNA. This mutation (a base substitution in the splicing sequence) eliminates a 3’ splice site resulting in the inclusion of the second intron (I2) in the final mRNA. We are going to pretend that this intron is short having only 15 nucleotides (most introns are much longer so this is just to make things simple) with the following sequence shown below in bold. The ( ) indicate the reading frames in the exons; the included intron 2 sequences are in bold. A. Would you expected this change to be harmful? ExplainB. If you were to do gene therapy to fix this problem, briefly explain what type of gene therapy youwould use to correct this. Please help. Thank youarrow_forward
- Molecular Biology Question Please help. Thank you Explain what is meant by the term “defective virus.” Explain how a defective virus is able to replicate.arrow_forwardMolecular Biology Explain why changing the codon GGG to GGA should not be harmful. Please help . Thank youarrow_forwardStage Percent Time in Hours Interphase .60 14.4 Prophase .20 4.8 Metaphase .10 2.4 Anaphase .06 1.44 Telophase .03 .72 Cytukinesis .01 .24 Can you summarize the results in the chart and explain which phases are faster and why the slower ones are slow?arrow_forward
- Can you circle a cell in the different stages of mitosis? 1.prophase 2.metaphase 3.anaphase 4.telophase 5.cytokinesisarrow_forwardWhich microbe does not live part of its lifecycle outside humans? A. Toxoplasma gondii B. Cytomegalovirus C. Francisella tularensis D. Plasmodium falciparum explain your answer thoroughly.arrow_forwardSelect all of the following that the ablation (knockout) or ectopoic expression (gain of function) of Hox can contribute to. Another set of wings in the fruit fly, duplication of fingernails, ectopic ears in mice, excess feathers in duck/quail chimeras, and homeosis of segment 2 to jaw in Hox2a mutantsarrow_forward
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