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You are a nurse caring for a patient with a pulmonary (lung) mold infection, The mold represents a large, macroscopic pathogen. Which of the following adaptive immune system responses would be MOST effective at controlling the mold infection? O High levels of IgA that promote neutralization O Hieh levels of leG that promotes agelutination, complement activation, and

You are a nurse caring for a patient with a pulmonary (lung) mold infection, The mold represents a large, macroscopic pathogen. Which of the following adaptive immune system responses would be MOST effective at controlling the mold infection? O High levels of IgA that promote neutralization O Hieh levels of leG that promotes agelutination, complement activation, and

Phlebotomy Essentials
Phlebotomy Essentials
6th Edition
ISBN:9781451194524
Author:Ruth McCall, Cathee M. Tankersley MT(ASCP)
Publisher:Ruth McCall, Cathee M. Tankersley MT(ASCP)
Chapter1: Phlebotomy: Past And Present And The Healthcare Setting
Section: Chapter Questions
Problem 1SRQ
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You are a nurse caring for a patient with a pulmonary (lung) mold infection, The mold represents a large, macroscopic pathogen. Which of the following adaptive immune system responses would be MOST effective at controlling the mold infection? O High levels of IgA that promote neutralization O High levels of IgG that promotes agglutination, complement activation, and opsonization An increase in host cell production of MHC I High levels of IgE that promote vascular change in association with mast cells O Production of a large population of antigen-specific Tc cells to destroy mold cells
Transcribed Image Text:You are a nurse caring for a patient with a pulmonary (lung) mold infection, The mold represents a large, macroscopic pathogen. Which of the following adaptive immune system responses would be MOST effective at controlling the mold infection? O High levels of IgA that promote neutralization O High levels of IgG that promotes agglutination, complement activation, and opsonization An increase in host cell production of MHC I High levels of IgE that promote vascular change in association with mast cells O Production of a large population of antigen-specific Tc cells to destroy mold cells
Hospital records show that a patient who died of MRSA strain 300 sepsis was infected two years ago with S. aureus (MRSA strain 500) and recovered after two weeks of antibiotic treatment. At the time of death, the patient had a large population of lymphocytes specific to S. aureus (MRSA strain 500). Which of the following explain WHY this population of specific lymphocytes failed to protect the patient: O None of the statements apply O The large population of lymphocytes specific to S. aureus (strain USA 500) were not responsive to IL-2 secretion The large population of memory cells specific to the antigens of S. aureus (MRSA strain 500) could not undergo primary response O The large population of memory cells specific to the antigens of S. aureus (MRSA strain 500) could not recognize antigens of S. aureus (MRSA strain 300) O The patient had no S. aureus (MRSA strain 300)-specific TH cells for the secretion of IL-2
Transcribed Image Text:Hospital records show that a patient who died of MRSA strain 300 sepsis was infected two years ago with S. aureus (MRSA strain 500) and recovered after two weeks of antibiotic treatment. At the time of death, the patient had a large population of lymphocytes specific to S. aureus (MRSA strain 500). Which of the following explain WHY this population of specific lymphocytes failed to protect the patient: O None of the statements apply O The large population of lymphocytes specific to S. aureus (strain USA 500) were not responsive to IL-2 secretion The large population of memory cells specific to the antigens of S. aureus (MRSA strain 500) could not undergo primary response O The large population of memory cells specific to the antigens of S. aureus (MRSA strain 500) could not recognize antigens of S. aureus (MRSA strain 300) O The patient had no S. aureus (MRSA strain 300)-specific TH cells for the secretion of IL-2
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