Gregor Mendel observed that some traits in his peas would disappear in the F1 generation but would then reappear in the next (F2) generation. He then proposed that A.new mutations were frequently generated in the F2 progeny, "reinventing" traits that had been lost in the F1. B.members of the F1 generation had only one allele for each character, but members of the F2 had two alleles for each character. C.the mechanism controlling the appearance of traits was different between the F1 and the F2 plants. D.this reappearance of the trait was due to dominance of an inherited factor over another. E.this was caused by a mutation that resulted in a dysfunctional protein being encoded by one of the two gene copies.

Name: Gregor Mendel observed that some traits in his peas would disappear in
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Description: Gregor Mendel observed that some traits in his peas would disappear in the F1 generation but would then reappear in the next (F2) generation. He then proposed that A.new mutations were frequently generated in the F2 progeny, "reinventing" traits that

Mendel observed by performing a cross between two pea plants having different traits that in the F1…

Answered: Gregor Mendel observed that some traits…

Human Anatomy & Physiology (11th Edition)

11th Edition

ISBN:9780134580999

Author:Elaine N. Marieb, Katja N. Hoehn

Publisher:Elaine N. Marieb, Katja N. Hoehn

Chapter1: The Human Body: An Orientation

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Gregor Mendel observed that some traits in his peas would disappear in the F1 generation but would then reappear in the next (F2) generation. He then proposed that

	A.	new mutations were frequently generated in the F2 progeny, "reinventing" traits that had been lost in the F1.
	B.	members of the F1 generation had only one allele for each character, but members of the F2 had two alleles for each character.
	C.	the mechanism controlling the appearance of traits was different between the F1 and the F2 plants.
	D.	this reappearance of the trait was due to dominance of an inherited factor over another.
	E.	this was caused by a mutation that resulted in a dysfunctional protein being encoded by one of the two gene copies.

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