During high-intensity exercise, why does arterial Pco2 decrease beyond its usual set point? Arterial Po2 is reduced during high intensity exercise, which stimulates chemoreceptors to increase ventilation beyond current CO2 production. O During high intensity exercise, the blood becomes more alkaline, which leads to more CO₂ being converted to H* and bicarbonate. Anaerobic metabolism does not produce CO2. O Acidic by-products of anaerobic metabolism decrease plasma pH, which stimulates central chemoreceptors to increase ventilation beyond current CO₂ production.

Human Anatomy & Physiology (11th Edition)
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ISBN:9780134580999
Author:Elaine N. Marieb, Katja N. Hoehn
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During high-intensity exercise, why does arterial Pco2 decrease beyond its usual set point?
Arterial Po₂ is reduced during high intensity exercise, which stimulates chemoreceptors to increase ventilation beyond current CO2 production.
During high intensity exercise, the blood becomes more alkaline, which leads to more CO2 being converted to H* and bicarbonate.
Anaerobic metabolism does not produce CO₂.
Acidic by-products of anaerobic metabolism decrease plasma pH, which stimulates central chemoreceptors to increase ventilation beyond
current CO₂ production.
Transcribed Image Text:During high-intensity exercise, why does arterial Pco2 decrease beyond its usual set point? Arterial Po₂ is reduced during high intensity exercise, which stimulates chemoreceptors to increase ventilation beyond current CO2 production. During high intensity exercise, the blood becomes more alkaline, which leads to more CO2 being converted to H* and bicarbonate. Anaerobic metabolism does not produce CO₂. Acidic by-products of anaerobic metabolism decrease plasma pH, which stimulates central chemoreceptors to increase ventilation beyond current CO₂ production.
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