Please help me with this question! Thank you!Please answer in no more than two sentence's for part A and part B. Thank you so much! **Cyanide blocks the mitochondrial electron transport chain. This inhibits ATP synthesis via oxidative phosphorylation and leads to death if someone ingests cyanide. Cyanide poisoning can be diagnosed when there are normal levels of O₂ but very low levels of NAD⁺.**a) Why do patients with cyanide poisoning have very low levels of NAD⁺? Answer in no more than two sentences.b) Patients with cyanide poisoning also display abnormally high levels of lactic acid in their cells. Why? Answer in no more than two sentences.

Cyanide poisoning is defined as the poisoning resulting from exposure to different forms of cyanide. Thus, it is a condition that will develop when a person will inhale, touch, or swallow cyanide. Cyanide is thus, a poisonous and harmful chemical gas that will prevent the human body from absorption of oxygen. The oxygen deficiency can further damage the organs.Cyanide binding to cytochrome a3 of electron transport chain (ETC) infers with the electrons acceptance from NADH as its power source, so NAD+ will not be able to regenerate, resulting in its low levels. Even when there is a normal oxygen availability, cells will not take or utilize oxygen from the bloodstream and lead to anaerobic respiration because oxygen is the final electron acceptor in the ETC. Cyanide will bind rapidly or instantly with cytochrome a3, a component of the cytochrome c oxidase complex present in mitochondria. Inhibition of cytochrome a3 will prevent or stop the cell from utilizing oxygen and, therefore, forces the use of anaerobic metabolism to result in lactic acid production. Thus, patients will have a high level of lactic acid.Thus, it can be concluded that cyanide poisoning causes oxygen and NAD+ deficiency and increases lactic acid production.