1. Excessive norepinephrine into the pre frontal cortex, activating the aplha 1 and beta adrenergic receptors, is most likely manifested in: * A.Productivity in expressive thought B.Profuse sweating, increased salivation, nausea C. Problems with working memory D. Positive symptoms of psychosis/schizophrenia (e.g. hallucinations)

Ciccarelli: Psychology_5 (5th Edition)
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Chapter1: The Science Of Psychology
Section: Chapter Questions
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PSYCHOLOGY

Choose the BEST answer for each item.
There is only one BEST answer so be sure to analyze and choose carefully.

1. Excessive norepinephrine into the pre frontal cortex, activating the aplha 1 and beta adrenergic receptors, is most likely manifested in: *

A.Productivity in expressive thought

B.Profuse sweating, increased salivation, nausea

C. Problems with working memory

D. Positive symptoms of psychosis/schizophrenia (e.g. hallucinations)
2. During the dreaming stage of sleep, serotonergic activity: *
A. Stays the same
B.Slows down and stops
C.Gradually speeds up and then peaks
D. Mediates the function of the adrenal glands
3. What is the effect of MAOs, SSRIs, SNRIs, & NERIs on synaptic transmission? *
 
A.Rapid enzymatic break down the neurotransmitter
B. Facilitate the synthesis and production of neurotransmitter
C. Allow the increased concentration of the neurotransmitter in the synaptic cleft
D. Enhance the storage capacity of the synaptic vesicles to hold more neurotransmitter molecules
4. What determines the effect of neurotransmitter release on the post synaptic neurons?
A. the size of the action potential
B. the post synaptic receptors
C. the size of the synaptic vesicles
D. the concentration of calcium ions reached in presynaptic terminals
E. the neurotransmitter contained in the synaptic vesicles
5. After an action potential has been created, and the neurotransmitter has been released to the synaptic cleft, presynaptic autoreceptors commonly
A. diffuse into the surrounding environment
B. depend on glial cells for the monitoring the neurotranmitter
C. initiate high affinity binding with ligands to send inhibitory post synaptic potentials
D. control further release of neurotransmitter to the synaptic cleft
6.The Striatal region/ Basal Ganglia is implicated in motor and purposeful movement. Which of the neurotransmitter/s is/are directly involved in this specific brain pathway and these motor behaviors? 
A. Dopamine
B. Serotonin
C.Norepinephrine
D. Dopamine and Serotonin
E. Serotonin and Norepinephrine
F. Dopamine and Norepinephrine
G. Dopamine, Serotonin, and Norepinephrine
H. None
7. Underactivity of dopamine will likely be manifested as: *
A. Ability to conceive children
B. Inability to stop consuming a chemical, drug, activity, or substance, even though it is causing psychological and physical harm.
C. Not able to do things
D.Perception of nonexistent object or event
8.Which of the following is TRUE about serotonin?
A. Binding of serotonin to COMT receptors creates excitatory effects.
B. Low serotonergic activity is linked to mood disorders like depression.
C. 5HT antagonists reduce feeling of hunger.
D. Melatonin is a precursor to serotonin.
9. Neurotransmitters can inhibit or excite neurons, depending on the receptor/s they bind with. ___ , for example, is inhibitory whereas ____ is excitatory. 
A.D1; D2
B. 5HT1a; 5HT2
C. Alpha 1 adrenergic; Beta adrenergic
D. None
10. ---- activity of ----- neutransmitter/s is/are implicated in Monoadrenergic Hypothesis of Depression.
A. over; Norepinephrine, Serotonin and Dopamine
B. over; Norepinephrine and Serotonin
C. over; Norepinephrine and Dopamine
D. over; Serotonin and Dopamine
E. under; Norepinephrine, Serotonin and Dopamine
F. under; Norepinephrine and Serotonin
G. under; Norepinephrine and Dopamine
H. under; Serotonin and Dopamine
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